63 Papers
107 Citations
Jun Han is an academic researcher from China Agricultural University. The author has contributed to research in topics: Porcine reproductive and respiratory syndrome virus & Virus. The author has an hindex of 20, co-authored 63 publications. Previous affiliations of Jun Han include University of Minnesota & Pennsylvania State University.
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Papers
Complete genome analysis of RFLP 184 isolates of porcine reproductive and respiratory syndrome virus.
Jun Han,Yue Wang,Kay S. Faaberg +2 more
TL;DR: The results suggest that targeted manipulation of PRRSV through nsp2 modification by reverse genetics may yield promising vectors for vaccine development, as has been recently demonstrated.
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Rangeland degradation and restoration management in China
TL;DR: In this paper, the importance of rangelands to China and its people is generally underestimated, legislative protection is incomplete and often ineffective, little attention is paid to the management of natural resources, and there is insufficient technological support, and Governments are not able to invest sufficiently to effectively restore and develop rangeland natural resources.
Pathogenesis and control of the Chinese highly pathogenic porcine reproductive and respiratory syndrome virus.
TL;DR: In this review, recent advances in understanding of the pathogenesis, evolution and ongoing field practices on the control of this troubling virus in China are summarized.
140
Function of glycoprotein E of herpes simplex virus requires coordinated assembly of three tegument proteins on its cytoplasmic tail.
TL;DR: It is found that the four proteins come together with very high efficiency to form a complex in transfected cells and in a manner that is regulated and coordinated.
107
Attenuation of porcine reproductive and respiratory syndrome virus strain MN184 using chimeric construction with vaccine sequence
Yue Wang,Yajie Liang,Jun Han,Kelly Burkhart,Eric Martin Vaughn,Michael B. Roof,Kay S. Faaberg +6 more
TL;DR: Chimeric virus treatment groups showed similar antibody responses as seen with these parent viruses, but much less severe pathogenesis when compared to the rMN184 group, suggesting that genetic aspects of Ingelvac PRRS MLV 5'UTR/ORF1 replicase region and/or the structural proteins/3'utR can serve to attenuate virulent strain MN184.
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