Jun Arita
Yokohama City University
31 Papers
538 Citations
Jun Arita is an academic researcher from Yokohama City University. The author has contributed to research in topics: Dopaminergic & Median eminence. The author has an hindex of 14, co-authored 31 publications.
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Papers
Loss of Estrogen-Induced Daily Surges of Prolactin and Gonadotropins by Suprachiasmatic Nucleus Lesions in Ovariectomized Rats*
TL;DR: In rats with large lesions of the suprachiasmatic nuclei (destruction of more than 50% of the bilateral nuclei), the characteristic PRL surges were eliminated, whereas comparablePRL surges to those in sham-lesioned anim...
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Effects of preoptic injections of gastrin, cholecystokinin, secretin, vasoactive intestinal peptide and PHI on the secretion of luteinizing hormone and prolactin in ovariectomized estrogen-primed rats.
TL;DR: The results suggest that brain-gut peptides act as neurotransmitters or neuromodulators in the preoptic region and are implicated in the regulation of LH and PRL secretion.
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Data on the Sites of Stimulatory Feedback Action of Gonadal Steroids Indispensable for Luteinizing Hormone Release in the Rat
TL;DR: The results suggest that the stimulatory feedback effect of E2 is exerted on the limbic structures, especially the BST and the l-SEPT, while the main sites of the Stimulatory feedback action of P are located in the DBB, the POSC, and the AHA.
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Identification by the sequential cell immunoblot assay of a subpopulation of rat dopamine-unresponsive lactotrophs.
TL;DR: The present study demonstrates that the sequential cell immunoblot assay is a useful means to quantify repeatedly hormone secretion from individual endocrine cells in culture and suggests that there is a subpopulation of rat PRL-secreting lactotrophs that are unresponsive to dopamine.
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Direct Inhibitory Effect of Long Term Estradiol Treatment on Dopamine Synthesis in Tuberoinfundibular Dopaminergic Neurons: In Vitro Studies Using Hypothalamic Slices
Jun Arita,Fukuko Kimura +1 more
TL;DR: The results suggest that chronically administered estradiol inhibits dopamine synthesis in TIDA neurons via a direct action on the hypothalamus and overcomes the facilitatory action of PRL on dopamine synthesis; and est radiol inhibits all three distinct systems that regulate basal, Ca2+-dependent, and cAMP-dependent dopamine synthesisation in Tida neurons.
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