Josef Arlt
University of Hamburg
14 Papers
224 Citations
Josef Arlt is an academic researcher from University of Hamburg. The author has contributed to research in topics: Schizophrenia (object-oriented programming) & Atrial natriuretic peptide. The author has an hindex of 7, co-authored 12 publications. Previous affiliations of Josef Arlt include Max Planck Society.
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Papers
Effects of a metabotropic glutamate(2/3) receptor agonist (LY544344/LY354740) on panic anxiety induced by cholecystokinin tetrapeptide in healthy humans: preliminary results.
Michael Kellner,Christoph Muhtz,Kristina Stark,Alexander Yassouridis,Josef Arlt,Klaus Wiedemann +5 more
TL;DR: A significant reduction of the number of CCK-4-induced panic symptoms and ofCCK- 4-induced subjective anxiety ratings was detected after removing two subjects who did not show decreased CCK -4-elicited adrenocorticotropin (ACTH) release after LY544344 compared to placebo treatment.
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The mineralocorticoid receptor agonist, fludrocortisone, inhibits pituitary-adrenal activity in humans after pre-treatment with metyrapone.
Christian Otte,Holger Jahn,Alexander Yassouridis,Josef Arlt,Nina Stober,Philip Maass,Klaus Wiedemann,Michael Kellner +7 more
TL;DR: Preliminary data suggest that MR are involved in the inhibition of the HPA axis during the circadian nadir of glucocorticoid concentrations in humans.
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Longitudinal course of salivary cortisol in post-traumatic stress disorder.
TL;DR: In chronic post‐traumatic stress disorder (PTSD) lowered cortisol secretion and hypersuppression to dexamethasone has been described repeatedly, but so far no longitudinal data on the natural course or on the effect of therapy are available.
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Adjunctive use of reboxetine in schizophrenia.
Thomas J. Raedler,Holger Jahn,Josef Arlt,Falk Kiefer,Mildred Schick,Dieter Naber,Klaus Wiedemann +6 more
TL;DR: The results suggest that the adjunctive use of reboxetine may be an effective treatment for depressive and negative symptoms in schizophrenia.
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Prolactin but not ACTH increases during sodium lactate-induced panic attacks
TL;DR: Prolactin release was significantly elevated in panickers compared with non-panickers, whereas ACTH secretion was not activated at all, although this differential stress response needs further investigation.
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