Jose R. Romero
Boston University
332 Papers
1.1K Citations
Jose R. Romero is an academic researcher from Boston University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 53, co-authored 268 publications. Previous affiliations of Jose R. Romero include University of Nebraska Medical Center & University of Nebraska–Lincoln.
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Papers
Digital Peripheral Arterial Tonometry and Cardiovascular Disease Events: The Framingham Heart Study.
Leroy L. Cooper,Na Wang,Alexa S. Beiser,Jose R. Romero,Hugo J. Aparicio,Vasileios-Arsenios Lioutas,Emelia J. Benjamin,Martin G. Larson,Ramachandran S. Vasan,Gary F. Mitchell,Sudha Seshadri,Sudha Seshadri,Naomi M. Hamburg +12 more
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Assessment of Risk Factors and Clinical Importance of Enlarged Perivascular Spaces by Whole-Brain Investigation in the Multi-Ethnic Study of Atherosclerosis
Sokratis Charisis,Tanweer Rashid,Hangfan Liu,Jeffrey B. Ware,Paul N. Jensen,Thomas R. Austin,Elyas Fadaee,Saima Hilal,Christopher Chen,Timothy M. Hughes,Jose R. Romero,Jon B. Toledo,W. T. Longstreth,Timothy J. Hohman,Ilya M. Nasrallah,Robert 'Nick' Bryan,Lenore J. Launer,Christos Davatzikos,Sudha Seshadri,Susan R. Heckbert,Mohamad Habes +20 more
TL;DR: In this article , a cross-sectional study was conducted in a multiethnic sample of 1026 individuals living in the community and the authors found that enlarged perivascular spaces in the basal ganglia and thalamus were associated with cerebral small-vessel disease.
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Aldosterone Modulates the Mechanistic Target of Rapamycin Signaling in Male Mice.
Danielle L Brooks,Amanda E Garza,Isis Akemi Katayama,Jose R. Romero,Gail K. Adler,Luminita H. Pojoga,Gordon H. Williams +6 more
TL;DR: Modulating mTOR activity may reduce aldosterone-dependent renal damage similar to mineralocorticoid receptor blockade but potentially with less adverse side effects.
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Spontaneous cervical spinal epidural hematoma mimicking acute stroke.
TL;DR: A case of spontaneous cervical spinal epidural hematoma (SEH) presenting with unilateral deficits mimicking acute stroke is presented.
Increased red cell sodium-lithium countertransport and lymphocyte cytosolic calcium are separate phenotypes in patients with essential hypertension
Jose R. Romero,Alicia Rivera,A Monari,Giulio Ceolotto,Andrea Semplicini,Paul R. Conlin,Paul R. Conlin +6 more
TL;DR: Elevated SLC activity and increased lymphocyte Cacyt are separate and distinct ion transport phenotypes in hypertensive patients, linked through a relationship to hyperinsulinaemia that is direct with S LC activity and inverse with lymphocyteCacyt.
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