Jordan Wright
University of Warwick
17 Papers
106 Citations
Jordan Wright is an academic researcher from University of Warwick. The author has contributed to research in topics: Lameness & Gene expression. The author has an hindex of 10, co-authored 16 publications. Previous affiliations of Jordan Wright include Oxford BioMedica & University of Oxford.
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Papers
Risk factors for reduced locomotion in dairy cattle on nineteen farms in The Netherlands.
TL;DR: The results provide a framework for hypotheses for future investigations of risk factors for high locomotion scores and identify factors associated with elevated mean locomotion score (increased abnormality) and the percentage of cows with the highest score.
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Management factors associated with impaired locomotion in dairy cows in England and Wales.
TL;DR: Putative risk factors support the hypothesis that locomotion score is linked to management factors; in particular, the combination of sawdust on rubber mats with automatic scrapers was associated with elevated locomotion scores.
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Promyelocytic Leukemia Protein Isoform II Promotes Transcription Factor Recruitment To Activate Interferon Beta and Interferon-Responsive Gene Expression
TL;DR: It is shown that PML isoform II (PML-II) is specifically required for efficient induction of IFN-β transcription and of numerous ISGs, acting at the point of transcriptional complex assembly on target gene promoters, and that in this way PML-II plays a significant role in the development of type I IFN responses.
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The Adenovirus E4-ORF3 Protein Stimulates SUMOylation of General Transcription Factor TFII-I to Direct Proteasomal Degradation
TL;DR: The results characterize a novel mechanism of TFII-I regulation by Ad and highlight how a viral protein can modulate a critical cellular transcription factor during infection.
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The Human Adenovirus 5 L4 Promoter Is Activated by Cellular Stress Response Protein p53
Jordan Wright,Keith N. Leppard +1 more
TL;DR: It is shown that p53 activation of L4P is significant during Ad5 infection, since depletion of p53 prior to infection of either immortalized or normal cells led to severely reduced late gene expression.
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