Jon S. Blevins
University of Arkansas for Medical Sciences
42 Papers
264 Citations
Jon S. Blevins is an academic researcher from University of Arkansas for Medical Sciences. The author has contributed to research in topics: Borrelia burgdorferi & Biology. The author has an hindex of 25, co-authored 41 publications. Previous affiliations of Jon S. Blevins include University of Texas Southwestern Medical Center.
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Papers
Mutation of sarA in Staphylococcus aureus limits biofilm formation.
TL;DR: In every strain other than Newman, including RN6390, simultaneous mutation of sarA and agr resulted in a phenotype like that observed with the sarA mutants, and implementation studies using a sarA clone confirmed that the defect in biofilm formation was due to the SarA mutation.
434
Multiplex PCR Protocol for the Diagnosis of Staphylococcal Infection
William J. Mason,Jon S. Blevins,Karen E. Beenken,Noroyono Wibowo,Neelum Ojha,Mark S. Smeltzer +5 more
TL;DR: The development of a multiplex PCR protocol for the diagnosis of staphylococcal infection was reported, which identified two isolates that were mecA positive but were oxacillin sensitive according to conventional methods.
255
Strain-Dependent Differences in the Regulatory Roles of sarA and agr in Staphylococcus aureus
Jon S. Blevins,Karen E. Beenken,Mohamed O. Elasri,Barry K. Hurlburt,Barry K. Hurlburt,Mark S. Smeltzer +5 more
TL;DR: It is suggested that studies defining the regulatory roles of sarA and agr by using RN6390 are not always representative of the events that occur in clinical isolates of S. aureus.
207
Staphylococcus aureus collagen adhesin contributes to the pathogenesis of osteomyelitis
Mohamed O. Elasri,J. R. Thomas,Robert A. Skinner,Jon S. Blevins,Karen E. Beenken,Carl L. Nelson,M.S Smelter +6 more
TL;DR: Results indicate that Cna is not required to establish joint infection, but does make an important contribution to the ability of S. aureus to establish infection in bone through hematogenous spread.
183
Analysis of the ospC Regulatory Element Controlled by the RpoN-RpoS Regulatory Pathway in Borrelia burgdorferi
Xiaofeng F. Yang,Meghan Lybecker,Utpal Pal,Sophie M. Alani,Jon S. Blevins,Andrew T. Revel,D. Scott Samuels,Michael V. Norgard +7 more
TL;DR: A body of evidence is provided that the RpoN-RpoS regulatory network controls ospC expression by direct binding of sigma(s) to a sigma (s)-dependent promoter of osp C.