John M. Davis
University of Illinois at Urbana–Champaign
31 Papers
275 Citations
John M. Davis is an academic researcher from University of Illinois at Urbana–Champaign. The author has contributed to research in topics: Schizophrenia & Reelin. The author has an hindex of 15, co-authored 31 publications. Previous affiliations of John M. Davis include East Tennessee State University.
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Papers
Embryonic development in the mouse is enhanced via microchannel culture
Stephanie Raty,E.M. Walters,John M. Davis,H.C. Zeringue,David J. Beebe,Sandra L. Rodriguez-Zas,Matthew B. Wheeler +6 more
TL;DR: The results suggest that the microchannel culture systems may provide a culture environment that more closely mimics the in vivo environment.
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Alterations in serum cortisol and its binding characteristics in anorexia nervosa.
TL;DR: Eight patients who were retested after weight gain, when they were between 76--100% of ideal body weight, exhibited no significant changes in serum steroids or in the binding capacity or apparent affinity of CBG for cortisol.
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Clinical Profile of an Atypical Antipsychotic: Risperidone
John M. Davis,Nancy Chen +1 more
TL;DR: R risperidone was consistently more effective than conventional antipsychotics in treating positive and negative symptoms and was effective in treating depression in schizophrenia.
Consistent evidence for a biological subtype of depression characterized by low CSF monoamine levels.
Robert D. Gibbons,John M. Davis +1 more
TL;DR: Maximum likelihood gaussian mixture analysis was used to resolve the empirical frequency distributions of both CSF HVA and CSF 5‐HIAA into two component normal mixtures, revealing a two component bivariate normal mixture distribution in which 35% of the depressed patient sample were classified in the low subgroup.
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Decrease in Reelin and Glutamic Acid Decarboxylase 67 (GAD 67 ) Expression in Schizophrenia and Bipolar Disorder
Alessandro Guidotti,James Auta,John M. Davis,Valeria DiGiorgi Gerevini,Yogesh Dwivedi,Dennis R. Grayson,Francesco Impagnatiello,Ghanshyam N. Pandey,Christine Pesold,Rajiv Sharma,D.P. Uzunov,Erminio Costa +11 more
- 01 Jan 2015
TL;DR: The selective down-regulation of RELN and GAD67 in prefrontal cortex of patients with schizophrenia and bipolar disorder who have psychosis is consistent with the hypothesis that these parameters are vulnerability factors in psychosis; this plus the loss of the correlation between these 2 parameters that exists in nonpsychotic subjects support the hypotheses that these changes may be liability factors underlying psychosis.
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