John G.F. Cleland
National Institutes of Health
1299 Papers
11.2K Citations
John G.F. Cleland is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Heart failure & Medicine. The author has an hindex of 137, co-authored 1172 publications. Previous affiliations of John G.F. Cleland include Northwestern University & Imperial College London.
Chat about Author
Papers
Measurement of troponin and natriuretic peptides shortly after admission in patients with heart failure-does it add useful prognostic information? An analysis of the Value of Endothelin Receptor Inhibition with Tezosentan in Acute heart failure Studies (VERITAS).
John G.F. Cleland,John G.F. Cleland,John R. Teerlink,Beth A. Davison,Ahmad Shoaib,Marco Metra,Stefanie Senger,Olga Milo,Gad Cotter,Robert C. Bourge,John D. Parker,Guillaume Jondeau,Henry Krum,Christopher M. O’Connor,Guillermo Torre-Amione,Dirk J. van Veldhuisen,John J.V. McMurray +16 more
TL;DR: This work aimed to investigate the added prognostic value of admission measurements of BNP and troponins in patients with acute heart failure.
32
Does aspirin attenuate the effect of angiotensin-converting enzyme inhibitors in hypertension or heart failure?
TL;DR: The evidence is not sufficient to justify advising long-term aspirin therapy for patients with cardiovascular disease in general and for those with heart failure in particular, and unless there is an opportunity to randomize the patient into a study of antithrombotic strategies, then aspirin should be withdrawn or possibly substituted with an anticoagulant or an antiplatelet agent that does not block cyclooxygenase.
32
Exploring quality of life in patients with and without heart failure
Jufen Zhang,James Hobkirk,Sean Carroll,Pierpaolo Pellicori,Andrew L. Clark,John G.F. Cleland +5 more
TL;DR: EHFSQ-1, comprises seven symptom clusters in patients with HF, and either summary factors or QoL scores can be used as an outcome measure, however, if the key question is 'what is this patient'sQoL?' rather than the reason why it is impaired, then a single, direct question may suffice.
32
Modification of atherosclerosis by agents that do not lower cholesterol
TL;DR: In patients who already have clinical evidence of coronary atherosclerosis there is now clear evidence that several different lipid lowering agents can delay or even reverse the atherosclerotic process reducing the incidence of cardiovascular events and delaying mortality.
31
Development and course of heart failure after a myocardial infarction in younger and older people.
TL;DR: The risk of developing HF and of dying after an MI increases progressively with age, Regardless of age, most deaths after a MI are preceded by the development of HF.
31