Jinying Luo
Fujian Medical University
13 Papers
6 Citations
Jinying Luo is an academic researcher from Fujian Medical University. The author has contributed to research in topics: Medicine & Gestational age. The author has an hindex of 3, co-authored 9 publications.
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Papers
Perinatal risk factors for congenital hypothyroidism: A retrospective cohort study performed at a tertiary hospital in China.
TL;DR: Perinatal factors should be considered during the diagnosis and treatment of CH, and significant associations were found with regard to parity and the risk of CH in the offspring.
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Risk factors of 125 cases of neonatal congenital hypothyroidism during perinatal period
Jinfu Zhou,Jinying Luo,Hong Zhao,Jing Wang,Feng Lin,Honghua Zhang,Yueqing Su,Yao Chen,Yinglin Zeng,Qingying Lin,Wenbin Zhu +10 more
TL;DR: CH was correlated with mother's age, gestational diabetes, Gestational thyroid disease as well as neonate's birth weight and gestational age, foetus number, fetal distress and other complicated birth defects at certain degree and more attention should be paid to perinatal care.
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Clinical characteristics of mirror syndrome: a retrospective study of 16 cases.
Ruiyun Chen,Min Liu,Jianying Yan,Fengyi Chen,Qing Han,Lianghui Zheng,Yulong Zhang,Jinying Luo,Lichun Chen,Hongfei Liu,Lu Yu +10 more
TL;DR: Elevated uric acid, lactate dehydrogenase, creatinine and D-dimer levels may be useful as predictors of mirror syndrome, which is not rare among patients with foetal hydrops.
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Association between genetic polymorphism of heme oxygenase 1 promoter and neonatal hyperbilirubinemia: a meta-analysis.
TL;DR: A statistically significant association was found between the HMOX1 (GT)n repeat length polymorphism and risk of neonatal hyperbilirubinemia under the allele and under the heterozygous genetic model.
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Expression of β-catenin in human trophoblast and its role in placenta accreta and placenta previa.
TL;DR: The expression of β-catenin in placenta accreta might play an important role in the regulation of placental cell invasion; low expression ofβ-catanin in Placenta Accretamight be responsible for excessive trophoblastic invasion.
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