Jinlong Wang
Jiangsu University
10 Papers
Jinlong Wang is an academic researcher from Jiangsu University. The author has contributed to research in topics: Melanoma & Glutaminase. The author has an hindex of 8, co-authored 9 publications.
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Papers
Long non-coding RNA HOTAIR acts as a competing endogenous RNA to promote malignant melanoma progression by sponging miR-152-3p
TL;DR: HOTAIR promotes the growth and metastasis of melanoma cells by competitively binding miR-152-3p, which functionally liberates c-MET mRNA and results in the activation of the downstream PI3k/Akt/mTOR signaling pathway.
Long noncoding RNA OIP5-AS1 acts as a competing endogenous RNA to promote glutamine catabolism and malignant melanoma growth by sponging miR-217.
TL;DR: It is demonstrated that OIP5‐AS1 acted as a sponge of miR‐217 to upregulate GLS expression, thus promoting glutamine catabolism and melanoma growth and provides a potential therapeutic target for patients with melanoma.
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miR-137 inhibits glutamine catabolism and growth of malignant melanoma by targeting glutaminase
TL;DR: It is shown that miR-137 was decreased in melanoma tissue, and the low mi R-137 level and high GLS expression are independent risk factor in melanomas, and it is demonstrated thatmiR- 137 acts as a tumor suppressor in melan cancer by targeting GLS.
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Exosomal miR-106b-5p derived from melanoma cell promotes primary melanocytes epithelial-mesenchymal transition through targeting EphA4
TL;DR: In this article, the role and molecular mechanisms of exosomal miR-106b-5p in melanoma remains unclear, however, it is shown that miR6b5p levels are significantly increased in patients with melanoma.
circRNA_0084043 promote malignant melanoma progression via miR-153-3p/Snail axis.
TL;DR: It is demonstrated that circRNA_0084043 may act as the sponge of miR-153-3p to up-regulate Snail expression, and consequently function as an oncogene in melanoma.