Jing Pan
Sichuan University
10 Papers
13 Citations
Jing Pan is an academic researcher from Sichuan University. The author has contributed to research in topics: Kidney & Medicine. The author has an hindex of 5, co-authored 8 publications.
Chat about Author
Papers
Understanding the gut-kidney axis among biopsy-proven diabetic nephropathy, type 2 diabetes mellitus and healthy controls: an analysis of the gut microbiota composition.
Sibei Tao,Lingzhi Li,Ling Li,Yuan Liu,Qian Ren,Min Shi,Jing Liu,Jing Jiang,Huichao Ma,Zhuo Huang,Zijing Xia,Jing Pan,Tiantian Wei,Yan Wang,Peiyun Li,Tian Lan,Xi Tang,Xiaoxi Zeng,Song Lei,Huairong Tang,Liang Ma,Liang Ma,Ping Fu +22 more
TL;DR: Gut microbiota composition was explored to be related to the occurrence of biopsy-proven DN from DM, and DM could be distinguished from HC by detecting g_Prevotella_9 level in feces, while DN was different from DM by the variables of g_Escherichia-Shigella and g_ Prevotella-9, which potentially contributed to the physiopathological diagnosis ofDN from DM.
134
Pterostilbene, a bioactive component of blueberries, alleviates renal fibrosis in a severe mouse model of hyperuricemic nephropathy
TL;DR: PTE suppressed the activation of TGF-β1/Smad3, Src and STAT3 signaling pathway to alleviate renal fibrosis of HN mice, highlighting that PTE was a potential antifibrotic strategy for hyperuricemic nephropathy.
49
Ethanol extract of Liriodendron chinense (Hemsl.) Sarg barks attenuates hyperuricemic nephropathy by inhibiting renal fibrosis and inflammation in mice.
Jing Pan,Chunle Zhang,Min Shi,Fan Guo,Jing Liu,Lingzhi Li,Qian Ren,Sibei Tao,Minghai Tang,Haoyu Ye,Liang Ma,Ping Fu +11 more
TL;DR: EELC alleviated the progression of HN by suppressing the activation of NF-κB, ASK1/JNK/c-Jun and JAK2/STAT3 signaling pathway, reducing the infiltration of inflammatory factors and uric acid accumulation in the kidney.
34
Pharmacologic inhibiting STAT3 delays the progression of kidney fibrosis in hyperuricemia-induced chronic kidney disease.
TL;DR: Wang et al. as mentioned in this paper used the mixture of adenine and potassium oxonate to perform kidney injury and fibrosis in hyperuricemic mice, accompanied by STAT3 activation in tubular and interstitial cells.
16
Toll-like Receptors as Potential Therapeutic Targets in Kidney Diseases.
TL;DR: New evidence is reviewed on roles of TLRs in the pathogenesis of kidney diseases including urinary tract infection, glomerulonephritis, acute kidney injury, transplant allograft dysfunction and chronic kidney diseases.
14