Jinfu Yang
Central South University
12 Papers
17 Citations
Jinfu Yang is an academic researcher from Central South University. The author has contributed to research in topics: NOX4 & Medicine. The author has an hindex of 4, co-authored 12 publications.
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Papers
Suppression of NADPH oxidase attenuates hypoxia-induced dysfunctions of endothelial progenitor cells
TL;DR: It is concluded that NOX-derived ROS contributes to the dysfunctions of EPCs under hypoxic condition and suppression of NOX may provide a novel strategy to improve endothelial functions in hypoxia-relevant diseases.
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Coordination between NADPH oxidase and vascular peroxidase 1 promotes dysfunctions of endothelial progenitor cells in hypoxia-induced pulmonary hypertensive rats.
E-Li Wang,Miao-Miao Jia,Fangmei Luo,Tao Li,Jing-Jie Peng,Xiu-Ju Luo,Feng-Lin Song,Jinfu Yang,Jun Peng,Bin Liu +9 more
TL;DR: It is concluded that NOX/VPO1 pathway-derived reactive oxygen species promote the oxidative injury and dysfunctions of EPCs in PH, which may contribute to endothelial Dysfunctions in PH.
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Magnesium Lithospermate B Derived from Salvia miltiorrhiza Ameliorates Right Ventricle Remodeling in Pulmonary Hypertensive Rats via Inhibition of NOX/VPO1 Pathway.
Tao Li,Jing-Jie Peng,E-Li Wang,Nian-Sheng Li,Feng-Lin Song,Jinfu Yang,Xiu-Ju Luo,Bin Liu,Jun Peng +8 more
TL;DR: It is concluded that MLB is able to prevent RV remodeling in hypoxic PAH rats through a mechanism involving a suppression of NOX/VPO1 pathway as well as ERK signaling pathway.
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Exosomes in atrial fibrillation: therapeutic potential and role as clinical biomarkers
TL;DR: In this paper, the role of exosomes and microRNAs in atrial fibrillation occurrence, their therapeutic potential, and their potential role as clinical biomarkers is considered.
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TMSB4 Overexpression Enhances the Potency of Marrow Mesenchymal Stromal Cells for Myocardial Repair.
Shiyuan Tang,Chengming Fan,Chukwuemeka Daniel Iroegbu,Wenwu Zhou,Zhigong Zhang,Ming Wu,Wangping Chen,Xiaoming Wu,Jun Peng,Zhihong Li,Jinfu Yang +10 more
TL;DR: In this paper, a rat model of myocardial infarction (MI) was used to identify the mechanism of HIF-1α induction by Tβ4 and investigate the effects of bone marrow mesenchymal stromal cells (BMMSCs) transfected with the Tβ 4 gene (TMSB4).
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