Jian Ping Zhou
Shanghai Jiao Tong University
17 Papers
12 Citations
Jian Ping Zhou is an academic researcher from Shanghai Jiao Tong University. The author has contributed to research in topics: Obstructive sleep apnea & Medicine. The author has an hindex of 4, co-authored 17 publications.
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Papers
Chronic intermittent hypoxia-induced mitochondrial dysfunction mediates endothelial injury via the TXNIP/NLRP3/IL-1β signaling pathway
Ya Ru Yan,Liu Zhang,Ying Ni Lin,Xian Wen Sun,Yong Jie Ding,Ning Li,Hong Peng Li,Shi Qi Li,Jian Ping Zhou,Qing Yun Li +9 more
TL;DR: In this article, the authors investigated the role of chronic intermittent hypoxia (CIH) induced mitochondrial dysfunction in vascular endothelial injury both in vivo and in vitro and found that the NOD-like receptor signaling pathway was involved in endothelial injuries induced by CIH, and showed that mito-TEMPO improved mitochondrial function and suppressed upregulation of the TXNIP/NLRP3/IL-1β pathway.
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The cumulative effect of air pollutants on the acute exacerbation of COPD in Shanghai, China.
TL;DR: Investigation of the cumulative effect of air pollutants on chronic obstructive pulmonary disease found high consecutive levels of PM2.5 and NO2 increase the risk of developing AECOPD and appears before the exacerbation onset.
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Impact of insomnia and obstructive sleep apnea on the risk of acute exacerbation of chronic obstructive pulmonary disease.
Shi Qi Li,Xian Wen Sun,Liu Zhang,Yong Jie Ding,Hong Peng Li,Ya Ru Yan,Ying Ni Lin,Jian Ping Zhou,Qing Yun Li +8 more
TL;DR: In this paper, sleep disorders are considered in clinical management for chronic obstructive pulmonary disease (COPD), and effective continuous positive airway pressure (CPAP) treatment may reduce the risk of AECOPD and mortality in patients with overlap syndrome.
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Angiotensin-(1-7) decreases the expression of collagen I via TGF-β1/Smad2/3 and subsequently inhibits fibroblast-myofibroblast transition.
TL;DR: The data suggests that Ang-(1-7) decreases the expression of Col-I via TGF-β1/Smad2/3 and subsequently inhibits HLF-MF transition.
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Intermittent Hypoxia Disrupts Glucose Homeostasis in Liver Cells in an Insulin-Dependent and Independent Manner.
Chen Juan Gu,Hua Hua Yi,Jing Feng,Zhiguo Zhang,Jun Zhou,Li Na Zhou,Jian Ping Zhou,Min Li,Qing Yun Li +8 more
TL;DR: IH in vitro impaired insulin signal transduction in liver cells as assessed by inhibited AKT/GSK-3β phosphorylation via JNK activation and JNK inhibitor SP600125.