Jesper Skovhus Thomsen
Aarhus University
203 Papers
1.4K Citations
Jesper Skovhus Thomsen is an academic researcher from Aarhus University. The author has contributed to research in topics: Bone remodeling & Medicine. The author has an hindex of 38, co-authored 190 publications. Previous affiliations of Jesper Skovhus Thomsen include Aarhus University Hospital & Copenhagen Business School.
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Papers
Morphology of the initial nephron-collecting duct connection in mice using computerized 3D tracing and electron microscopy.
Ping Zhang,Ling Gu,Jing Cong,Jie Zhang,Jesper Skovhus Thomsen,Arne Andreasen,Shi-Jie Chang,Si-Qi Deng,Jia Xing,Xiao-Yue Zhai +9 more
TL;DR: The present study demonstrated that the initial connection was composed of cells which were closely associated with the renal vesicle, the initial nephron, and connected with the basal epithelium of the terminal UB tip at discrete points.
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Animal models of disuse-induced bone loss: study protocol for a systematic review.
TL;DR: This systematic review will provide an overview of available animal models of disuse-induced bone loss and discuss the different methods used to quantify and analyze the bone loss.
2D size of trabecular bone structure units (BSU) correlate more strongly with 3D architectural parameters than age in human vertebrae.
Britney Alexi Lamarche,Jesper Skovhus Thomsen,Christina Møller Andreasen,W. Brent Lievers,Thomas Levin Andersen +4 more
TL;DR: In this paper , the authors focused on the recently formed trabecular bone structural unit (BSU) and how their 2D size and shape changes with age and bone microstructure.
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No influence of alimentary zinc on the healing of calvarial defects filled with osteopromotive substances in rats
TL;DR: Findings indicated that healing of adult rat calvarial defects is not influenced by alimentary zinc supplementation or depletion, and defects filled with demineralized bone matrix were significantly stronger and exhibited significantly more new bone formation than defectsfilled with autogenous bone or unfilled controls.
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Modeling-based bone formation transforms trabeculae to cortical bone in the sclerotic areas in Buschke-Ollendorff syndrome. A case study of two females with LEMD3 variants.
Morten Frost,Morten Frost,Morten Frost,E T Rahbek,Charlotte Ejersted,Poul Flemming Høilund-Carlsen,Poul Flemming Høilund-Carlsen,Anette Bygum,Anette Bygum,Jesper Skovhus Thomsen,Christina Møller Andreasen,Christina Møller Andreasen,Thomas Levin Andersen,Anja Lisbeth Frederiksen,Anja Lisbeth Frederiksen +14 more
TL;DR: While bone turnover and BMD were largely within normal reference range in patients with the Buschke-Ollendorff syndrome, osteosclerotic lesions appear to emerge due to modeling-based bone formation with secondary bone remodeling.
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