Jerome E. Granato
University of Virginia
7 Papers
170 Citations
Jerome E. Granato is an academic researcher from University of Virginia. The author has contributed to research in topics: Hemodynamics & Dipyridamole. The author has an hindex of 5, co-authored 7 publications.
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Papers
Utility of two-dimensional echocardiography in suspected ascending aortic dissection
TL;DR: 2-D echo represents a reliable noninvasive method for rapidly diagnosing ascending aortic dissection at the bedside; offers important prognostic information which is directly related to the presence of pericardial fluid, and provides useful additional information which assists or establishes an alternative diagnosis when ascending aortal dissection is absent.
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Myocardial thallium-201 kinetics during coronary occlusion and reperfusion: influence of method of reflow and timing of thallium-201 administration.
TL;DR: Thallium-201 (201Tl) uptake and redistribution kinetics were examined in an open-chest canine preparation of occlusion and reperfusion and Microsphere-determined transmural blood flow was similar in all groups during LAD occlusions and final flows after 2 hr were comparable in all subgroups undergoing reflow.
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Effects of dipyridamole and aminophylline on hemodynamics, regional myocardial blood flow and thallium-201 washout in the setting of a critical coronary stenosis
TL;DR: In a canine model of partial coronary stenosis, systemic hypotension, adverse regional flow effects (coronary steal) and prolonged thallium-201 washout consequent to intravenously administered dipyridamole are promptly reversed by intravenous aminophylline administration.
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Myocardial thallium-201 kinetics and regional flow alterations with 3 hours of coronary occlusion and either rapid reperfusion through a totally patent vessel or slow reperfusion through a critical stenosis.
TL;DR: 3 hours of coronary occlusion followed by either rapid reperfusion through a totally patent vessel or slow reperfusions through a critical stenosis resulted in little delayed thallium-201 redistribution or myocardial salvage as assessed histologically, despite significant recovery of regional flow.
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A unique chromosomal abnormality in idiopathic hypereosinophilic syndrome presenting with cardiac involvement.
TL;DR: The patient's MVP diminished, until finally it was no longer evident on the M-mode views and was barely detectable on the long-axis 2D views, in all likelihood caused by aging and distension of the mitral subvalvular apparatus or anulus.
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