Jamal Benhamida
University of California, San Francisco
4 Papers
2 Citations
Jamal Benhamida is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Melanoma & Cancer. The author has an hindex of 3, co-authored 4 publications.
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Papers
Genomic and Transcriptomic Analysis Reveals Incremental Disruption of Key Signaling Pathways during Melanoma Evolution.
A. Hunter Shain,Nancy M. Joseph,Richard Yu,Jamal Benhamida,Shanshan Liu,Tarl W. Prow,Beth S. Ruben,Beth S. Ruben,Jeffrey P. North,Laura B. Pincus,Iwei Yeh,Robert L. Judson,Boris C. Bastian +12 more
TL;DR: Genomic and transcriptomic changes that accompany the evolution of melanoma from pre-malignant lesions are elucidated by sequencing DNA and RNA from primary melanomas and their adjacent precursors, as well as matched primary tumors and regional metastases.
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Pericytic tumors of the kidney—a clinicopathologic analysis of 17 cases
Deepika Sirohi,Steven C. Smith,Jonathan I. Epstein,Bonnie Balzer,Jeffry P. Simko,Dana Balitzer,Jamal Benhamida,Oleksandr N. Kryvenko,Nilesh S. Gupta,Swetha Paluru,Isabela Werneck da Cunha,Daniel N. Leal,Sean R. Williamson,Mariza de Peralta-Venturina,Mahul B. Amin,Mahul B. Amin +15 more
TL;DR: The series further corroborates that malignant behavior is rare, and that criteria associated with aggression among soft tissue pericytic tumors may not be predictive for those in the kidney, and identifies a wide morphologic spectrum, including lesions presenting composite morphologies.
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Mesenteric inflammatory veno-occlusive disease: radiographic and histopathologic evaluation of 2 cases
TL;DR: Two cases of histologically proven MIVOD in patients presenting with abdominal pain are reported and potentially useful distinguishing features at contrast-enhanced CT are described, including prominent small pericolonic arteries and veins but a diminutive or absent inferior mesenteric vein.
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Abstract 2372: The genetic evolution of melanoma
Alan Hunter Shain,Richard Yu,Iwei Yeh,Jamal Benhamida,Ivanka Kovalyshyn,Aravindhan Sriharan,Eric Talevich,Reinhard Dummer,Jeffrey P. North,Laura B. Pincus,Beth S. Ruben,William Rickaby,Corrado D’Arrigo,Alistair Robson,Robert L. Judson,Nancy M. Joseph,Boris C. Bastian +16 more
TL;DR: A framework of the genetic evolution of melanocytic neoplasm from precursor lesions is outlined and the rate-limiting homeostatic factors that become disrupted as neoplasms progress from one stage to the next are unveiled, as are the pathogenic factors driving this evolution.
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