J. Tao
5 Papers
J. Tao is an academic researcher. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 2, co-authored 3 publications.
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Papers
Changes in dorsal root ganglion CGRP expression in mouse pinch nerve injury model: Modulation by Somatostatin type-2 receptor
TL;DR: In this paper , the authors examined pain behavior and the expression of neuropeptides such as calcitonin gene-related peptide (CGRP) in dorsal root ganglion neurons(DRGs) as well as the changes of the number of CGRP-IR DRGs in the mouse model of sciatic pinch nerve injury.
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Grain-sized moxibustion at Zusanli (ST36) promotes hepatic autophagy in rats with hyperlipidemia by regulating the ULK1 and TFEB expression through the AMPK/mTOR signaling pathway
TL;DR: Grain-sized moxibustion at ST36 acupoints could regulate the blood lipid level of Sprague-Dawley (SD) rats with hyperlipidemia, increase the expression level of ULK1 and TFEB by activating the AMPK/mTOR signaling pathway in liver tissues, and initiate the transcription of autophagy genes such as LC3 as mentioned in this paper .
A Review of Gap Junction Protein and its Potential Role in Nervous System-Related Disease.
Qiong Xiang,Xiao-Lin Liu,Jia-Jia Chen,Liang Yang,Li-Ni Liu,Jing Deng,J. Tao,Xian-Hui Li +7 more
TL;DR: The possible mechanism of its action in nervous system-related diseases and treatment are reviewed and its function of regulating immune response, cell proliferation, migration, apoptosis, and carcinogenesis is reviewed.
The integrated analysis and underlying mechanisms of FNDC5 on diabetic induced cognitive deficits.
Qiong Xiang,J. Tao,Chuan-Jun Fu,Li-Xiu Liao,Li-Ni Liu,Jing Deng,Xian-Hui Li +6 more
TL;DR: The current study revealed that the synaptic plasticity of hippocampal cells in hyperglycemia might be regulated by FNDC5\BDNF-Trk axis, playing the protective role in the process of CI induced by hyperglycemia and providing a target for the early treatment of hyperglycemia induced cognitive dysfunction in clinic.
What is the role of Von Willebrand factor in chronic hepatitis B virus infection to hepatocellular carcinoma: a review article
TL;DR: In this article , the role of von Willebrand factor (VWF) in the development of hepatocellular carcinoma (HCC) and the functional domain of VWF in cancer as well as cross-talking with platelets and miRNAs.