J Scott
University of Liverpool
11 Papers
147 Citations
J Scott is an academic researcher from University of Liverpool. The author has contributed to research in topics: Langerhans cell & Neck dissection. The author has an hindex of 7, co-authored 11 publications.
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Papers
•Journal Article
Pathological findings in clinically false-negative and false-positive neck dissections for oral carcinoma.
TL;DR: It is concluded that the most stringent clinical protocols, even when supplemented by CT scanning, cannot be expected to achieve 100% accuracy in diagnosing cervical metastatic disease.
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A histological analysis of the early effects of alcohol and tobacco usage on human lingual epithelium.
TL;DR: The structural changes appeared to be non-specific reactions to local toxic effects of alcohol and tobacco, which could indicate an increased vulnerability to carcinogens, whether or not these derive from alcoholic drinks or tobacco smoke.
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Histological analysis of parotid and submandibular glands in chronic alcohol abuse: a necropsy study.
J Scott,J Burns,E A Flower +2 more
TL;DR: Neither grossly detectable parotid enlargement nor acinar hypertrophy, a feature which has previously been noted as characteristic of alcoholic sialadenosis, were evident in this series, which provide little structural support for the reportedly increased secretory capacity of salivary glands in chronic alcohol abuse.
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Effect of tobacco and alcohol consumption on the Langerhans cell population of human lingual epithelium determined using a monoclonal antibody against HLADR.
TL;DR: The results suggest that either human oral LC respond to external toxins, or that physical changes in the epithelial barrier induced by such toxins necessitate the presence of more LC to maintain the integrity of the epithelium.
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Morphometric analysis of atrophic changes in human lingual epithelium in iron deficiency anaemia.
TL;DR: A stereological analysis of epithelial structure at the lateral surface of the tongue showed that iron deficiency anaemia was associated with reduced epithelial thickness despite the absence of overt mucosal abnormalities, which may be a trophic response to the overall loss of epithelium in this condition.
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