J. R. Goding
University of Melbourne
37 Papers
1K Citations
J. R. Goding is an academic researcher from University of Melbourne. The author has contributed to research in topics: Aldosterone & Luteinizing hormone. The author has an hindex of 21, co-authored 37 publications. Previous affiliations of J. R. Goding include Worcester Foundation for Biomedical Research.
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Papers
Prostaglandin F2α identified as a Luteolytic Hormone in Sheep
John A. McCracken,J. C. Carlson,M. E. Glew,J. R. Goding,J. R. Goding,David T. Baird,David T. Baird,K. Green,B. Samuelsson +8 more
TL;DR: Prostaglandin F2α is released from the uterus of the sheep in a cyclic fashion, acts primarily in a local manner on the ovary via a counter-current mechanism and is responsible for the periodic regression of the corpus luteum in this species.
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The control of aldosterone secretion.
TL;DR: There is also evidence that increased secretion of aldosterone is a contributory cause of oedema occurring in kidney, liver and heart disease as discussed by the authors, however, this is not a characteristic sign in the aldosteron secreting tumours which were first described by Conn.
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Constancy of Interval Between Luteinizing Hormone Release and Ovulation in the Ewe
TL;DR: The constancy of the interval between LH release and ovulation in these experiments confirms the importance of the role of LH in the ovulatory physiology.
A heterologous radioimmunoassay for follicle stimulating hormone: application to measurement of FSH in the ovine estrous cycle, and in several other species including man.
Lois A. Salamonsen,Helen A. Jonas,H. G. Burger,Joan M. Buckmaster,Chamley Wa,I. A. Cumming,Jock K. Findlay,J. R. Goding +7 more
TL;DR: A heterologous radioimmunoassay system, using an antiserum to human FSH in combination with radioiodinated ovine FSH was highly specific for FSH for a number of species, and was used to measure plasma FSH levels in sheep, rat and man in a range of physiological states.
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Control of Adrenal Secretion of Electrolyte-active Steroids—I
TL;DR: The finding that there is increased urinary excretion of aldosterone in congestive cardiac failure as in other oedematous states (Chart and Shipley, 1953) has raised the question whether increased adrenal cortical activity is a contributory cause of the development of oedema.
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