http://xoomer.virgilio.it/lusebra/NuoviFile/PrevenzioneTVP.pdf

Prevention of venous thromboembolism

Venous thromboembolism (VTE) is categorized by the U.S. Surgeon General as a major public health problem. VTE is relatively common and associated with reduced survival and substantial health-care costs, and recurs frequently. VTE is a complex (multifactorial) disease, involving interactions between acquired or inherited predispositions to thrombosis and VTE risk factors, including increasing patient age and obesity, hospitalization for surgery or acute illness, nursing-home confinement, active cancer, trauma or fracture, immobility or leg paresis, superficial vein thrombosis, and, in women, pregnancy and puerperium, oral contraception, and hormone therapy. Although independent VTE risk factors and predictors of VTE recurrence have been identified, and effective primary and secondary prophylaxis is available, the occurrence of VTE seems to be relatively constant, or even increasing.

/pdf/the-epidemiology-of-venous-thromboembolism-5d9tzjofn1.pdf

The epidemiology of venous thromboembolism

https://journal.chestnet.org/article/S0012-3692(08)60117-8/pdf

Pharmacology and management of the vitamin K antagonists: American College of Chest Physicians Evidence-Based Clinical Practice Guidelines (8th Edition).

Oral Anticoagulants: Mechanism of Action, Clinical Effectiveness, and Optimal Therapeutic Range

https://scholarlypublications.universiteitleiden.nl/access/item%3A3137579/view

Venous thrombosis: a multicausal disease.

La necrose de la peau est une complication rare des anticoagulants oraux. Une observation confirme le role propose de la deficience de proteine C dans le mecanisme de la necrose de la peau, au stade initial du traitement anticoagulant oral. Elle montre de plus que les coumariniques peuvent etre mal toleres chez les malades avec une forme grave de deficience congenitale de proteine C. L'administration combinee de doses progressivement croissantes d'un anticoagulant oral d'action prolongee et d'heparine a des doses therapeutiques pourrait entrainer une anticoagulation stable sans complication

Successful progressive anticoagulation in a severe protein C deficiency and previous skin necrosis at the initiation of oral anticoagulant treatment.

Modified apc resistance assay for patients on oral anticoagulants

Homozygous protein C deficiency with late onset and recurrent coumarin-induced skin necrosis.

Effects of long term administration of ticlopidine on platelet function and hemostatic variables.

Risk of thrombosis in patients homozygous for factor V Leiden [letter; comment]

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