Immacolata Cacace
Sapienza University of Rome
8 Papers
6 Citations
Immacolata Cacace is an academic researcher from Sapienza University of Rome. The author has contributed to research in topics: Hypnosis & Somatosensory evoked potential. The author has an hindex of 7, co-authored 8 publications.
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Papers
Perception and modulation of pain in waking and hypnosis: functional significance of phase-ordered gamma oscillations.
TL;DR: High and medium hypnotizable subjects showed significant reductions in phase‐ordered gamma patterns for Focused Analgesia during hypnosis and post‐hypnosis conditions; this effect was found, however, more pronounced in high hypnotized subjects, suggesting that hypnosis interferes with phase‐ ordered gamma and pain relationship.
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Neuroticism-Anxiety, Impulsive-Sensation Seeking and autonomic responses to somatosensory stimuli
TL;DR: This pattern of findings was taken to suggest that high Imp-SS participants are more arousable and less prone to defensive reactions to novel or aversive stimulation.
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Pain perception, obstructive imagery and phase-ordered gamma oscillations.
TL;DR: Evidence is provided for the role of gamma oscillations in the subjective experience of pain and for the view that pain reduction during obstructive mental imagery is the product of an inhibitory process involving frontal and parietal cortical regions.
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Hypnotizability-related EEG alpha and theta activities during visual and somesthetic imageries.
Francesca I. Cavallaro,Immacolata Cacace,Massimiliano Del Testa,Paolo Andre,Giancarlo Carli,Vilfredo De Pascalis,Raffaele Rocchi,Enrica L. Santarcangelo +7 more
TL;DR: The results indicate that different, hypnotizability-related cognitive strategies, that are revealed by differences in EEG modulation, are responsible for the similar subjective experience associated with visual and somesthetic imageries in Highs and Lows.
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Pain Modulation in Waking and Hypnosis in Women: Event-Related Potentials and Sources of Cortical Activity
TL;DR: It is demonstrated that hypnotic suggestions can exert a top-down modulatory effect on attention/preconscious brain processes involved in pain perception by reducing the strength of the association of pain modulation and brain activity changes at BA3.