Involvement of cannabinoid CB2 receptors in the IgE-mediated triphasic cutaneous reaction in mice.

Kampo formulations and allergic inflammatory diseases

https://www.jstage.jst.go.jp/article/jtm/26/4/26_4_141/_pdf

To elucidate individual pathogenic alteration (so-called "Sho") diagnosed by Kampo medicine -Evidence-based objective evaluation of the efficacy of Kampo formulations-

Previous　studies　have　reported　that　the　mice　passively　sensitized　with　anti－DNP　IgE　antibody exhibited　IgRmediated　biphasic　cutaneous　reaction　with　an　immediate　phase　response（IPR）at　l　h　and a　late　phase　response（LPR）at24h　after　the　challenge　of　DNFB（dinitrofluorobenzene）．We　recently found　that　the　third　phase　inflammatory　response　with　intense　and　persisting　infiltration　of　eosinophils， named　very　late　phase　response（vLPR），was　induced　following　IPR　and　LPR　in　response　to　DNFB　in passively　sensitized　mice，and　that　the　peak　response　of　vLPR　was　on　the8th　day　after　the　challenge． The　inhibitory　effect　of　Kampo　medicines　on　the　triphasic　cutaneous　inflammatory　reaction　was　divided into　several　groups　in　terms　of　their　inhibition　rate　of　ear　swelling．Among　the　formulations，Tokaku－ joki－to（Tao－He－Cheng－Qi－Tang）was　effective　at　inhibiting　IPR，LPR　and　vLPR（＋／＋／＋group） and　scratching　behavior　in　IPR．The　Inhibitory　effect　of　Tokaku－joki－to　on　triphasic　cutaneous　reaction primarily　depends　on　its　composed　cmde　dmgs，Glycyrrhizae　Radix　and　Cimamomi　Cortex．These

Effect of Some Kampo Medicines, Including Tokaku-joki-to(Tao-He-Cheng-Qi-Tang), on IgE-mediated Triphasic Skin Reaction in Passively Sensitized Mice.

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Effect of Some Kampo Medicines, Including Tokaku-joki-to(Tao-He-Cheng-Qi-Tang), on IgE-mediated Triphasic Skin Reaction in Passively Sensitized Mice.