Huiling Yang
Guangdong Medical College
20 Papers
157 Citations
Huiling Yang is an academic researcher from Guangdong Medical College. The author has contributed to research in topics: Nasopharyngeal carcinoma & Nasopharyngeal neoplasm. The author has an hindex of 13, co-authored 19 publications. Previous affiliations of Huiling Yang include University of South China & Southern Medical University.
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Papers
Tumor suppressor PDCD4 modulates miR-184- mediated direct suppression of C-MYC and BCL2 blocking cell growth and survival in nasopharyngeal carcinoma
Yan Zhen,Zhen Liu,Zhen Liu,Huiling Yang,Huiling Yang,Xiaoli Yu,Qiangyun Wu,Shengni Hua,Xiaobin Long,Qingping Jiang,Ye-Qiong Song,Chao Cheng,Hao Hao Wang,M Zhao,Qiaofen Fu,Xiaoming Lyu,Yiyu Chen,Yue-Qin Fan,Yan Liu,Xin Li,Weiyi Fang +20 more
TL;DR: These studies are the first to demonstrate that PDCD4 as tumor suppressor regulated miR-184-mediated direct targeting of BCL2 and C-MYC via PI3K/AKT and JNK/C-Jun pathway attenuating cell proliferation and survival in NPC.
Loss of connective tissue growth factor as an unfavorable prognosis factor activates miR-18b by PI3K/AKT/C-Jun and C-Myc and promotes cell growth in nasopharyngeal carcinoma.
Xi-Yong Yu,Yan Zhen,Huiling Yang,Huizhen Wang,Yangshu Zhou,E Wang,Francesco M. Marincola,C Mai,Yuyao Chen,H Wei,Ye Song,Xiaoming Lyu,Yongyi Ye,L Cai,Qiangyun Wu,M Zhao,Shengni Hua,Qiaofen Fu,Y Zhang,Kaitai Yao,Zhile Liu,Xuenong Li,Weiyi Fang +22 more
TL;DR: These studies are the first to demonstrate that reduced CTGF as an unfavorable prognosis factor mediates the activation of miR-18b, an oncomir directly suppresses CTGF expression, by PI3K/AKT/C-Jun and C-Myc and promotes cell growth of NPC.
Caveolin-1 enhances resveratrol-mediated cytotoxicity and transport in a hepatocellular carcinoma model.
Huiling Yang,Wei-qiong Chen,Xuan Cao,Andrea Worschech,Andrea Worschech,Andrea Worschech,Li-fen Du,Weiyi Fang,Yang-Yan Xu,David F. Stroncek,Xin Li,Ena Wang,Francesco M. Marincola +12 more
TL;DR: It is observed that Caveolin-1(CAV1) expression can increase the cytotoxic and pro-apoptotic activity of RES in a dose- and time-dependent manner both in vitro and in vivo in a Hepatocellular Carcinoma animal model.
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Loss of cytoplasmic KLF4 expression is correlated with the progression and poor prognosis of nasopharyngeal carcinoma.
Zhen Liu,Zhen Liu,Huiling Yang,Huiling Yang,Weiren Luo,Weiren Luo,Qingping Jiang,Chunping Mai,Yan Chen,Yan Zhen,Xiaoli Yu,Xiaobin Long,Weiyi Fang +12 more
TL;DR: In nasopharyngeal carcinoma (NPC), the correlation of Krüppel‐like factor 4 (KLF4) expression with clinicopathological features including patient prognosis is examined.
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Reduced CTGF Expression Promotes Cell Growth, Migration, and Invasion in Nasopharyngeal Carcinoma
Yan Zhen,Yanfen Ye,Xiaoli Yu,Chunping Mai,Ying Zhou,Yan Chen,Huiling Yang,Xiaoming Lyu,Ye Song,Qiangyun Wu,Qiaofen Fu,Mengyang Zhao,Shengni Hua,Hao Wang,Zhen Liu,Yajie Zhang,Weiyi Fang +16 more
TL;DR: This study demonstrates that reduced expression of CTGF promoted cell proliferation, migration, invasion and cell cycle progression through FAK/PI3K/AKT, EMT and MMP pathways in NPC.