15 Papers
12 Citations
Hui He is an academic researcher from Dalian Medical University. The author has contributed to research in topics: Oncogene & Cell cycle. The author has an hindex of 9, co-authored 14 publications. Previous affiliations of Hui He include China Medical University (PRC).
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Papers
Lysine-specific demethylase 5C promotes hepatocellular carcinoma cell invasion through inhibition BMP7 expression.
TL;DR: In this article, KDM5C is abundantly expressed in invasive human hepatocellular carcinoma (HCC) cells and ectopic expression in HCC cells promoted cell migration, invasion and epithelial-mesenchymal transition via inactivation of BMP7.
The fate of Krüppel-like factor 9-positive hepatic carcinoma cells may be determined by the programmed cell death protein 5
TL;DR: It is confirmed that the levels of KLF9 mRNA and protein were lower in hepatocellular carcinoma (HCC) tissue compared to normal tissue and that PDCD5 overexpression stimulated the promoter activities of KLf9 by luciferase reporter assays.
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Recombinant human PDCD5 exhibits an antitumor role in hepatocellular carcinoma cells via clathrin‑dependent endocytosis
TL;DR: The anti-tumor effect of rhPDCD5 in HCC cells is mediated via CDE, and it was confirmed that the pre‑operative serum levels of PDCD5 protein in patients with HCC were significantly lower than the post-operative serum Levels.
Review of 43 patients with autoimmune pancreatitis based on the international consensus diagnostic criteria in China.
TL;DR: The aims of this study were to systematically analyze and identify clinical features of the largest cohort of patients with AIP from a single institution in China, and to identify patients who fulfilled the International Consensus Diagnostic Criteria for AIP.
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HOXD9 promotes epithelial-mesenchymal transition and cancer metastasis by ZEB1 regulation in hepatocellular carcinoma.
Xiupeng Lv,Linlin Li,L i Lv,Xiaotong Qu,Shi Jin,Kejun Li,Xiaoqin Deng,Lei Cheng,Hui He,Lei Dong +9 more
TL;DR: Hepatocellular carcinoma overexpression can significantly enhance HCC cell migration, invasion, and metastasis, whereas silencing HOXD9 inhibits these processes.