Hui He
Southwest University
10 Papers
Hui He is an academic researcher from Southwest University. The author has contributed to research in topics: Apoptosis & Viability assay. The author has an hindex of 6, co-authored 10 publications.
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Papers
Sensitization of tamoxifen-resistant breast cancer cells by Z-ligustilide through inhibiting autophagy and accumulating DNA damages
TL;DR: Findings provide important insights into the formation of tamoxifen resistance in breast cancer cells, but also suggest Z-ligustilide may function as a novel autophagy inhibitor to overcome chemoresistance.
Ginsenoside 20(S)-Rh2 Induces Apoptosis and Differentiation of Acute Myeloid Leukemia Cells: Role of Orphan Nuclear Receptor Nur77.
Chengqiang Wang,Hui He,Guojun Dou,Juan Li,Xiao-Mei Zhang,Mingdong Jiang,Pan Li,Xiaobo Huang,Hongxi Chen,Li Li,Da-Jian Yang,Hongyi Qi +11 more
TL;DR: The studies suggest that the Nur77-mediated signaling pathway is highly involved in 20(S)-Rh2-induced apoptosis and differentiation of AML cells.
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Z-ligustilide restores tamoxifen sensitivity of ERa negative breast cancer cells by reversing MTA1/IFI16/HDACs complex mediated epigenetic repression of ERa.
TL;DR: It is shown, for the first time, that Z-ligustilide restored the growth inhibition of tamoxifen on ERα− breast cancer cells, and opens new possibilities from TCM for treating aggressive tamoxIFen-resistant breast cancer.
Isobavachalcone inhibits acute myeloid leukemia: Potential role for ROS-dependent mitochondrial apoptosis and differentiation.
TL;DR: Isobachalcone (IBC) has been shown to induce apoptosis and differentiation of acute myeloid leukemia (AML) cells, but the underlying molecular mechanisms are not fully understood as mentioned in this paper.
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β-Asarone Induces Apoptosis and Cell Cycle Arrest of Human Glioma U251 Cells via Suppression of HnRNP A2/B1-Mediated Pathway In Vitro and In Vivo.
Li Li,Yi Yang,Mingxia Wu,Zanyang Yu,Chengqiang Wang,Guojun Dou,Hui He,Hongmei Wang,Na Yang,Hongyi Qi,Xiaoyu Xu +10 more
TL;DR: In this paper, the authors showed that β-asarone, the main component in the volatile oil of Acori tatarinowii rhizoma, inhibited the cell viability, proliferation, and colony formation ability of U251 cells.
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