Hugo A. Katus
Heidelberg University
1553 Papers
9.9K Citations
Hugo A. Katus is an academic researcher from Heidelberg University. The author has contributed to research in topics: Medicine & Myocardial infarction. The author has an hindex of 107, co-authored 1496 publications. Previous affiliations of Hugo A. Katus include University Hospital Heidelberg.
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Papers
Usefulness of percutaneous aortic valve implantation to improve quality of life in patients >80 years of age.
Raffi Bekeredjian,Ulrike Krumsdorf,Emanuel Chorianopoulos,Klaus Kallenbach,Mathias Karck,Hugo A. Katus,Wolfgang Rottbauer +6 more
TL;DR: A significant clinical benefit from TAVI is shown in a patient population aged ≥81 years, and both the physical and the mental component summary scores improved significantly.
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Effects of the main green tea polyphenol epigallocatechin-3-gallate on cardiac involvement in patients with AL amyloidosis
TL;DR: Consumption of green tea polyphenol EGCG in patients with cardiac involvement with AL amyloidosis causes a significant decrease in left ventricular wall thickness and mass, as well as an improvement in NYHA functional classification andleft ventricular ejection fraction.
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Improved outcomes after heart transplantation for cardiac amyloidosis in the modern era.
Arnt V. Kristen,Michael M. Kreusser,Patrick Blum,Stefan Schönland,Lutz Frankenstein,Andreas Dösch,Benjamin Knop,Matthias Helmschrott,Bastian Schmack,Arjang Ruhparwar,Ute Hegenbart,Hugo A. Katus,Philip Raake +12 more
TL;DR: It is concluded that heart transplantation in highly selected patients with isolated non-systemic advanced cardiac amyloidosis may be a feasible approach.
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Two Novel Members of the ABLIM Protein Family, ABLIM-2 and -3, Associate With STARS and Directly Bind F-actin
Tomasa Barrientos,Derk Frank,Koichiro Kuwahara,Svetlana Bezprozvannaya,G. C. Teg Pipes,Rhonda S Bassel-Duby,James A. Richardson,Hugo A. Katus,Eric N. Olson,Norbert Frey +9 more
TL;DR: It is suggested that the members of the novel ABLIM protein family may serve as a scaffold for signaling modules of the actin cytoskeleton and thereby modulate transcription.
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C1q-TNF-Related Protein-9 Promotes Cardiac Hypertrophy and Failure.
Mahesh Appari,Astrid Breitbart,Florian Brandes,Malgorzata Szaroszyk,Natali Froese,Mortimer Korf-Klingebiel,Mona Malek Mohammadi,Andrea Grund,Gesine M. Scharf,Honghui Wang,Carolin Zwadlo,Daniela Fraccarollo,Ulrike Schrameck,Mona Nemer,G. William Wong,Hugo A. Katus,Kai C. Wollert,Oliver J. Müller,Johann Bauersachs,Joerg Heineke +19 more
TL;DR: The upregulation of CTRP9 during hypertrophic heart disease facilitates maladaptive cardiac remodeling and left ventricular dysfunction and might constitute a therapeutic target in the future.
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