Huanjun Wang
Shandong University
12 Papers
43 Citations
Huanjun Wang is an academic researcher from Shandong University. The author has contributed to research in topics: Thyroid cancer & Metastasis. The author has an hindex of 3, co-authored 11 publications.
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Papers
Nevirapine Increases Sodium/Iodide Symporter-Mediated Radioiodide Uptake by Activation of TSHR/cAMP/CREB/PAX8 Signaling Pathway in Dedifferentiated Thyroid Cancer.
TL;DR: Results indicated that nevirapine significantly inhibited the proliferation and increased the expressions of thyroid differentiation-related genes, thyroid stimulating hormone receptor (TSHR), sodium/iodide symporter (NIS), thyroid peroxidase (TPO), and transcriptional factor paired box 8 (PAX8) in dedifferentiated thyroid cancer cells.
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•Journal Article
Association between chemokine receptor 5 (CCR5) delta32 gene variant and atherosclerosis: a meta-analysis of 13 studies.
TL;DR: These studies add to the evidence that CCR5 Δ32-positive genotype (Δ32/Δ 32 or wt/ Δ32) increases the risk of atherosclerotic disease in Asian population.
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Hypercholesterolemia Is an Associated Factor for Risk of Differentiated Thyroid Cancer in Chinese Population.
TL;DR: Wang et al. as mentioned in this paper investigated the relationship between hypercholesterolemia and differentiated thyroid cancer (DTC) in Chinese population and found that the closer the serum total cholesterol level is to normal, the less the risk of thyroid nodules being malignant is, and this difference is statistically significant.
•Journal Article
Association between sodium iodide symporter and differentiated Thyroid cancer: a meta-analysis of 9 studies.
Rui Zhang,Huanjun Wang,Junyu Zhao,Jinming Yao,Hongxia Shang,Huangao Zhu,Lin Liao,Jianjun Dong +7 more
TL;DR: The expression of NIS is significantly increased in DTC, which could help explain the reason for individual with a poor response to RAI therapy, and the reduced iodide uptake in thyroid cancer may not caused by the decreased expression ofNIS, function of N IS protein or its post-transcriptional translocation.
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Pituitary resistance to thyroid hormone caused by a novel mutation (H435A) in the thyroid hormone receptor beta: A case report.
Changzhen Yu,Junyu Zhao,Jinming Yao,Huanjun Wang,Hongxia Shang,Rui Zhang,Yujiao Cui,Li-kang Wang,Jianjun Dong,Liao Lin +9 more
TL;DR: A new mutation (H435A) of the thyroid hormone receptor beta gene was found, which allowed for the establishment of a definitive diagnosis of pituitary resistance to thyroid hormones, and the outcome in the reported case has been satisfactory despite the persistence of non-suppressed TSH.
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