Hong Peng Li
Shanghai Jiao Tong University
13 Papers
6 Citations
Hong Peng Li is an academic researcher from Shanghai Jiao Tong University. The author has contributed to research in topics: Medicine & Obstructive sleep apnea. The author has an hindex of 3, co-authored 9 publications.
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Papers
Chronic intermittent hypoxia-induced mitochondrial dysfunction mediates endothelial injury via the TXNIP/NLRP3/IL-1β signaling pathway
Ya Ru Yan,Liu Zhang,Ying Ni Lin,Xian Wen Sun,Yong Jie Ding,Ning Li,Hong Peng Li,Shi Qi Li,Jian Ping Zhou,Qing Yun Li +9 more
TL;DR: In this article, the authors investigated the role of chronic intermittent hypoxia (CIH) induced mitochondrial dysfunction in vascular endothelial injury both in vivo and in vitro and found that the NOD-like receptor signaling pathway was involved in endothelial injuries induced by CIH, and showed that mito-TEMPO improved mitochondrial function and suppressed upregulation of the TXNIP/NLRP3/IL-1β pathway.
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Nocturnal Mean Oxygen Saturation Is Associated with Secondary Polycythemia in Young Adults with Obstructive Sleep Apnea, Especially in Men.
TL;DR: Nocturnal mean SpO2 was an independent predictor of polycythemia in young adults and Hemoglobin and hematocrit values might have diagnostic utility for assessing nocturnal hypoxia severity of OSA patients, especially in men.
Impact of insomnia and obstructive sleep apnea on the risk of acute exacerbation of chronic obstructive pulmonary disease.
Shi Qi Li,Xian Wen Sun,Liu Zhang,Yong Jie Ding,Hong Peng Li,Ya Ru Yan,Ying Ni Lin,Jian Ping Zhou,Qing Yun Li +8 more
TL;DR: In this paper, sleep disorders are considered in clinical management for chronic obstructive pulmonary disease (COPD), and effective continuous positive airway pressure (CPAP) treatment may reduce the risk of AECOPD and mortality in patients with overlap syndrome.
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Bronchial Variation: Anatomical Abnormality May Predispose Chronic Obstructive Pulmonary Disease.
TL;DR: In this article, the authors identified three common types of bronchial variation that were used to formulate a unifying hypothesis to explain how these variations contribute to the development of chronic obstructive pulmonary disease (COPD).
Surfaxin attenuates PM2.5-induced airway inflammation via restoring surfactant proteins in rats exposed to cigarette smoke.
Xian Wen Sun,Ying Ni Lin,Yong Jie Ding,Shi Qi Li,Hong Peng Li,Jian Ping Zhou,Liu Zhang,Ji Min Shen,Qing Yun Li +8 more
TL;DR: In this article, the authors showed that Surfaxin protects the alveolar epithelium from PM2.5 in airway inflammation through increasing Surfactant proteins (SPs) including SPA, SPB, SPC and SPD.
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