Hong Li
Tongji University
40 Papers
111 Citations
Hong Li is an academic researcher from Tongji University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 11, co-authored 22 publications.
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Papers
Association between metabolic syndrome and osteoporosis: A meta-analysis
Jiaojiao Zhou,Jiaojiao Zhou,Qiang Zhang,Qiang Zhang,Xiaolan Yuan,Xiaolan Yuan,Jijiao Wang,Chunyan Li,Chunyan Li,Hui Sheng,Shen Qu,Hong Li +11 more
TL;DR: The findings suggest that MetS is a risk factor for developing osteoporosis in men and a negative effect of MetS on BMD in men but not in women.
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Eosinophils Reduce Chronic Inflammation in Adipose Tissue by Secreting Th2 Cytokines and Promoting M2 Macrophages Polarization
TL;DR: The role of eosinophils in regulating metabolic homeostasis and obesity is focused on.
Elucidation of the Intestinal Absorption Mechanism of Celastrol Using the Caco-2 Cell Transwell Model.
Hong Li,Jie Li,Lu Liu,Yichuan Zhang,Yili Luo,Xiaoli Zhang,Peng Yang,Manna Zhang,Weifeng Yu,Shen Qu +9 more
TL;DR: The intestinal transport of celastrol was a time- and concentration-dependent active transport, and the efflux of celastsrol was energy dependent, indicating that celastro is a substrate of P-glycoprotein but not multidrug resistance protein 2 or the breast cancer resistance protein.
Reversibility of β-Cell-Specific Transcript Factors Expression by Long-Term Caloric Restriction in db/db Mouse
TL;DR: The work showed that the course of β-cell dedifferentiation can intervene by long-term control of calorie intake, and key β- cell-specific TFs and functional factors play important roles in maintainingβ-cell differentiation.
Differential pattern for regulating insulin secretion, insulin resistance, and lipid metabolism by osteocalcin in male and female T2DM patients.
Xue-Fei Rui,Bei Xu,Junlei Su,Chunping Pan,Chenyu Zhan,Bin Su,Hong Li,Jiying Wang,Hui Sheng,Shen Qu +9 more
TL;DR: Osteocalcin might improve glucose metabolism through enhancing insulin secretion in males, and through increasing insulin secretion and improving insulin resistance in females with T2DM.