Heejung Jun
UPRRP College of Natural Sciences
5 Papers
37 Citations
Heejung Jun is an academic researcher from UPRRP College of Natural Sciences. The author has contributed to research in topics: Synaptic plasticity & Ccaat-enhancer-binding proteins. The author has an hindex of 5, co-authored 5 publications.
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Papers
SALM Synaptic Cell Adhesion-like Molecules Regulate the Differentiation of Excitatory Synapses
Jaewon Ko,Seho Kim,Hye Sun Chung,Karam Kim,Kihoon Han,Hyun Kim,Heejung Jun,Bong-Kiun Kaang,Eunjoon Kim +8 more
TL;DR: The identification of a family of cell adhesion-like molecules termed SALM that interacts with the abundant postsynaptic density (PSD) protein PSD-95 suggests that SALM2 is an important regulator of the differentiation of excitatory synapses.
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Regulation of ApC/EBP mRNA by the Aplysia AU-rich element-binding protein, ApELAV, and its effects on 5-hydroxytryptamine-induced long-term facilitation.
Se‐Jeong Yim,Yongseok Lee,Jin-A Lee,Deok-Jin Chang,Jin-Hee Han,Hyoung F. Kim,Hyungju Park,Heejung Jun,V. Narry Kim,Bong-Kiun Kaang +9 more
TL;DR: The results imply that the Aplysia ARE‐binding protein, ApELAV, can regulate ApC/EBP gene expression at the mRNA level, and accordingly, ARE‐mediated post‐transcriptional mechanism may serve a crucial function in regulating the expression of ApC /EBP in response to a 5‐HT stimulus.
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AU-rich element-binding protein negatively regulates CCAAT enhancer-binding protein mRNA stability during long-term synaptic plasticity in Aplysia
Yongseok Lee,Sun Lim Choi,Heejung Jun,Se Jeong Yim,Jin-A Lee,Hyoung F. Kim,Seung-Hee Lee,Jaehoon Shim,Kyungmin Lee,Deok-Jin Jang,Bong-Kiun Kaang +10 more
TL;DR: Evidence is provided for a critical role of the posttranscriptional modification of ApC/EBP mRNA during the consolidation of synaptic plasticity in Aplysia by cloned an AU-rich element (ARE)-binding protein, ApAUF1, which functions as a destabilizing factor for ApC or EBP mRNA.
14
Synaptic adhesion molecule IgSF11 regulates synaptic transmission and plasticity
Seil Jang,Daeyoung Oh,Yeunkum Lee,Eric Hosy,Hyewon Shin,Christoph Van Riesen,Daniel J. Whitcomb,Julia M. Warburton,Jihoon Jo,Jihoon Jo,Doyoun Kim,Sun-Gyun Kim,Seung Min Um,Seok-Kyu Kwon,Myoung Hwan Kim,Myoung Hwan Kim,Junyeop Daniel Roh,Jooyeon Woo,Heejung Jun,Dongmin Lee,Won Mah,Hyun Kim,Bong-Kiun Kaang,Kwangwook Cho,Jeong-Seop Rhee,Daniel Choquet,Eunjoon Kim +26 more
TL;DR: The results suggest that IgSF11 regulates excitatory synaptic transmission and plasticity through its tripartite interactions with PSD-95 and AMPARs.
PKA-activated ApAF–ApC/EBP heterodimer is a key downstream effector of ApCREB and is necessary and sufficient for the consolidation of long-term facilitation
Jin-A Lee,Sue-Hyun Lee,Changhoon Lee,Deok-Jin Chang,Yong Lee,Hyoung F. Kim,Ye-Hwang Cheang,Hyoung-Gon Ko,Yongseok Lee,Heejung Jun,Dusan Bartsch,Eric R. Kandel,Bong-Kiun Kaang +12 more
TL;DR: It is suggested that PKA-activated ApAF–ApC/EBP heterodimer is a core downstream effector of ApCREB in the consolidation of LTF.