Harald Fritz
University of Jena
26 Papers
221 Citations
Harald Fritz is an academic researcher from University of Jena. The author has contributed to research in topics: Hypothermia & Traumatic brain injury. The author has an hindex of 14, co-authored 26 publications. Previous affiliations of Harald Fritz include Schiller International University.
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Papers
Effect of Empirical Treatment With Moxifloxacin and Meropenem vs Meropenem on Sepsis-Related Organ Dysfunction in Patients With Severe Sepsis: A Randomized Trial
Frank M. Brunkhorst,Michael Oppert,Gernot Marx,Frank Bloos,Katrin Ludewig,Christian Putensen,Axel Nierhaus,Ulrich Jaschinski,Andreas Meier-Hellmann,Andreas Weyland,Matthias Gründling,Onnen Moerer,Reimer Riessen,Armin Seibel,Maximilian Ragaller,Markus W. Büchler,Stefan John,Friedhelm Bach,Claudia Spies,Lorenz Reill,Harald Fritz,Michael Kiehntopf,Evelyn Kuhnt,Holger Bogatsch,Christoph Engel,Markus Loeffler,Marin H. Kollef,Konrad Reinhart,Tobias Welte +28 more
TL;DR: In this paper, the authors compared the effect of moxifloxacin and meropenem with the effect on sepsis-related organ dysfunction and concluded that meropenems alone did not result in less organ failure.
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The effect of mild hypothermia on plasma fentanyl concentration and biotransformation in juvenile pigs.
TL;DR: A prolonged increase of the plasma fentanyl concentration should be anticipated for appropriate control of the analgesia/sedatives during and early after therapeutic hypothermia.
101
A comparison between meperidine, clonidine and urapidil in the treatment of postanesthetic shivering.
TL;DR: Meperidine and clonidine were both nearly 100% effective in treating postanesthetic shivering without negative side effects, whereas urapidil was only effective in 60% of patients treated.
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Coupling of cerebral blood flow and oxygen metabolism in infant pigs during selective brain hypothermia
TL;DR: The data demonstrate that SBH decreases CBF and oxygen metabolism to a degree comparable with the cerebrovascular and metabolic effects of systemic hypothermia, and cerebral metabolism is a main determinant of CBF during SBH.
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A pig model with secondary increase of intracranial pressure after severe traumatic brain injury and temporary blood loss.
TL;DR: An animal model of severe TBI with a reproducible secondary ICP increase accompanied by patterns of diffuse brain damage is described and may be helpful in the study of pathogenetic relevance of concomitant affections and verify new therapeutic approaches insevere TBI.
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