Hanbin Cui
Ningbo University
16 Papers
1 Citations
Hanbin Cui is an academic researcher from Ningbo University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 3, co-authored 3 publications.
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Papers
Genome-Wide Association and Functional Studies Identify SCML4 and THSD7A as Novel Susceptibility Genes for Coronary Artery Disease.
Yang Li,Dao Wen Wang,Yundai Chen,Can Chen,Jian Guo,Shu Zhang,Zhijun Sun,Hu Ding,Yan Yao,Lei Zhou,Ke Xu,Chun Song,Fan Yang,Bin Zhao,Han Yan,Wen-Jing Wang,Chong Wu,Xiangfeng Lu,Xueli Yang,Jie Dong,Gu-Yan Zheng,Shuhan Tian,Yanjun Cui,Li-Juan Jin,Gangqiong Liu,Hanbin Cui,Shenghuang Wang,Feng Jiang,Chang-Hua Wang,Jeanette Erdmann,Linyao Zeng,Shian Huang,Jianfeng Zhong,Yuehua Ma,Wenjiang Chen,Jianli Sun,Wei Lei,Shenghan Chen,Shaoqi Rao,Dongfeng Gu,Heribert Schunkert,Xiao-Li Tian +41 more
TL;DR: It is shown that 2 genes, SCML4 and THSD7A, make functional contributions to atherosclerosis and how rs852787 and its host gene DAB1 are linked to CAD needs further studies.
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Common genetic variants of the β2-adrenergic receptor affect its translational efficiency and are associated with human longevity
Ling Zhao,Fan Yang,Ke Xu,Huiqing Cao,Gu Yan Zheng,Zhang Yan,Jianxin Li,Hanbin Cui,Xiaomin Chen,Zhiming Zhu,Hongbo He,Xianming Mo,Brian K. Kennedy,Yousin Suh,Yi Zeng,Yi Zeng,Xiao-Li Tian +16 more
TL;DR: The data indicate that enhanced production of β2‐adrenergic receptors caused by genetic variants is inversely associated with human lifespan.
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Serum Urate and Heart Failure: A Bidirectional Mendelian Randomisation Study.
Fangkun Yang,Teng Hu,Hanbin Cui +2 more
TL;DR: Consistent evidence is provided for the causal effect of genetically predicted serum urate level on HF, but not the reverse effect of HF, and urate-lowering therapy may be of cardiovascular benefit in the prevention of HF.
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Microbiota‐related metabolites fueling the understanding of ischemic heart disease
TL;DR: A recent review as mentioned in this paper summarized recent advances in the IHD-linked alterations in microbiome, not only with a focus on the taxa of bacteria, but also with a particular emphasis on the microbiota-related metabolites that regulate the initiation, escalation, and onset of IHD.
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