Han Sung Jung
Yonsei University
254 Papers
1.3K Citations
Han Sung Jung is an academic researcher from Yonsei University. The author has contributed to research in topics: Biology & Medicine. The author has an hindex of 37, co-authored 229 publications. Previous affiliations of Han Sung Jung include Guangzhou Medical University & Harvard University.
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Papers
The role of angiogenesis and pulpal healing in tooth replantation and allograft transplantation.
TL;DR: In this paper, the expression patterns of revascularization and pulpal healing, which are the most important for the pulp viability, were analyzed after tooth replantation and allograft in mice.
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Retinoic acid modulates chondrogenesis in the developing mouse cranial base.
Hyuk Jae Kwon,Jeong Oh Shin,Jong Min Lee,Kyoung Won Cho,Min Jung Lee,Sung Won Cho,Han Sung Jung +6 more
TL;DR: It is demonstrated that RA modulates chondrocytes to proliferate, differentiate, or undergo programmed cell death during endochondral bone formation in the developing cranial base.
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Gastrin-releasing peptide expression and its effect on the calcification of developing mouse incisor
TL;DR: It is suggested that GRP plays a significant role in the formation of enamel and dentin by regulating ameloblasts and predentin formation, respectively, and GRP signaling is strongly related to calcium acquisition and secretion during mouse incisor development.
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Use of Tethered Hydrogel Microcoatings for Mesenchymal Stem Cell Equilibrium, Differentiation, and Self‐Organization into Microtissues
David W. Green,David W. Green,Gregory S. Watson,Jolanta A. Watson,Jong Min Lee,Han Sung Jung,Han Sung Jung +6 more
- 16 Nov 2017
TL;DR: It is shown that a transient ultrathin covering of permeable biomaterial can be differentially formulated to either preserve multipotency or induce multidifferentiation in therapeutic adult mesenchymal stem cells (MSCs).
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Ameloblastoma modifies tumor microenvironment for enhancing invasiveness by altering collagen alignment
TL;DR: Ameloblastoma-mediated ECM remodeling contributes to the formation of an invasive collagen architecture during tumor progression and shows the capacity to remodel the ECM independently of cancer-associated fibroblasts.
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