Göran Källner
Karolinska University Hospital
30 Papers
412 Citations
Göran Källner is an academic researcher from Karolinska University Hospital. The author has contributed to research in topics: Atrial fibrillation & Calcitonin gene-related peptide. The author has an hindex of 16, co-authored 29 publications. Previous affiliations of Göran Källner include Karolinska Institutet.
Chat about Author
Papers
Effects of intramyocardial injection of phVEGF-A165 as sole therapy in patients with refractory coronary artery disease – 12-month follow-up: Angiogenic gene therapy
Nondita Sarkar,Andreas Rück,Göran Källner,Shams Y-Hassan,Pontus Blomberg,Khalid B. Islam,J. van der Linden,Dan Lindblom,Anders T. Nygren,B Lind,Lars-Åke Brodin,Viktor Drvota,Christer Sylvén +12 more
TL;DR: Effects of intramyocardial injection of phVEGF‐A165 as sole therapy in patients with refractory coronary artery disease: 12‐month follow‐up.
79
Myocardial Injury After Electrical Therapy for Cardiac Arrhythmias Assessed by Troponin-T Release
TL;DR: Episodes of ventricular fibrillation with subsequent intracardiac, and to a lesser extent, external defibrillation give rise to a statistically significant increase in S-troponin T, S-CK-MB(mass) and S-myoglobin indicative of a minor myocardial injury or dysfunction.
68
Capsazepine-sensitive release of calcitonin gene-related peptide from C-fibre afferents in the guinea-pig heart by low pH and lactic acid
TL;DR: The use of capsazepine to inhibit the function of C-fibre afferents may represent a novel principle to influence autonomic reflex reactions associated with cardiac pathological conditions.
65
Release and effects of calcitonin gene-related peptide in myocardial ischaemia.
TL;DR: It may be suggested that local cardiac CGRP-release from capsaicin-sensitive C-fibre afferents during myocardial ischaemia functions as an endogenous physiological protective response.
62
Aggravation of Myocardial Infarction in the Porcine Heart by Capsaicin-induced Depletion of Calcitonin Gene-related Peptide (cgrp)
TL;DR: This study indicates that peptides released from cardiac C fibers have a beneficial effect in myocardial ischemia and reperfusion and is a possible candidate for the mediation of the observed cardioprotective effect.
52