Glenn D. Prestwich
University of Utah
693 Papers
11.2K Citations
Glenn D. Prestwich is an academic researcher from University of Utah. The author has contributed to research in topics: Self-healing hydrogels & Lysophosphatidic acid. The author has an hindex of 88, co-authored 690 publications. Previous affiliations of Glenn D. Prestwich include Stony Brook University & University of Tennessee.
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Papers
Phosphatidylinositol 4,5‐bisphosphate regulates inspiratory burst activity in the neonatal mouse preBötzinger complex
Erin A. Crowder,Margaret S. Saha,Ryland W. Pace,Honglu Zhang,Glenn D. Prestwich,Christopher A. Del Negro +5 more
TL;DR: It is proposed that PIP2 protects ICAN from rundown by interacting directly with underlying ion channels and preventing desensitization, which may enhance the robustness of respiratory rhythm.
Heparin-regulated release of growth factors in vitro and angiogenic response in vivo to implanted hyaluronan hydrogels containing VEGF and bFGF
Daniel B. Pike,Shenshen Cai,Kyle R. Pomraning,Matthew A. Firpo,Robert J. Fisher,Xiao Zheng Shu,Glenn D. Prestwich,Robert A. Peattie +7 more
TL;DR: The ability to stimulate localized microvessel growth at controlled rates for extended times through the release of GFs from covalently linked, Hp-supplemented hydrogels will ultimately provide a powerful therapeutic tool.
Injectable glycosaminoglycan hydrogels for controlled release of human basic fibroblast growth factor
TL;DR: Covalently linked, heparin-containing glycosaminoglycan hydrogels that can be injected and crosslinked in situ constitute highly promising new materials for controlled release of hepar in-binding growth factors in vivo.
Dual growth factor-induced angiogenesis in vivo using hyaluronan hydrogel implants.
Robert A. Peattie,Erin R. Rieke,Erin M. Hewett,Robert J. Fisher,Xiao Zheng Shu,Glenn D. Prestwich +5 more
TL;DR: New therapeutic approaches for numerous pathologies could be notably enhanced by the localized, sustained angiogenic response produced by release of both VEGF and KGF from crosslinked HA films.
Prevention of anti-microbial peptide LL-37-induced apoptosis and ATP release in the urinary bladder by a modified glycosaminoglycan.
Won Yong Lee,Justin R. Savage,Jianxing Zhang,Wanjian Jia,Siam Oottamasathien,Glenn D. Prestwich +5 more
TL;DR: It is suggested that GM-0111 and possibly GAG molecules prevent the development of cystitis by blocking the apoptosis and the concurrent release of ATP from the urothelium.