Ge Yang
Changchun University
10 Papers
34 Citations
Ge Yang is an academic researcher from Changchun University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 3, co-authored 5 publications.
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Papers
Relationship between endothelial nitric oxide synthase gene polymorphisms and ischemic stroke: a meta-analysis.
TL;DR: A meta‐analysis of studies examining whether polymorphisms in the endothelial nitric oxide synthase (eNOS) gene are associated with ischemic stroke risk found no association.
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Compromised cerebrovascular modulation in chronic anxiety: evidence from cerebral blood flow velocity measured by transcranial Doppler sonography
TL;DR: The results indicate that cerebrovascular modulation is compromised in chronic anxiety; anxious subjects have some insufficiency in maintaining cerebral perfusion after postural change.
1044P Taxanes plus immunotherapy might be a potential option for HER2-altered NSCLC beyond first-line progression: A retrospective real-world study
TL;DR: In this article , the authors compared the efficacy of taxanes-based chemotherapy alone (C), and combined with angiogenesis inhibitors (C+A) or immune checkpoint inhibitors(C+I) for advanced non-small cell lung cancer (NSCLC).
2
Using CT imaging features to predict visceral pleural invasion of non-small-cell lung cancer.
Y. Yang,Z. Xie,H Hu,Ge Yang,Xi Zhu,D. Yang,Zhongxi Niu,Guangxian Mao,Miner Shao,J. Wang +9 more
TL;DR: The image feature model showed great potential in predicting pleural invasiveness, and had comparable diagnostic efficacy compared with the combined model containing clinical data.
2
Xenon-enhanced CT assessment of cerebral blood flow in stroke-in-progress patients with unilateral internal carotid artery or middle cerebral artery stenosis.
TL;DR: Xe-CT was used to examine cerebral perfusion in patients with or without SIP and found infarctions were surrounded by larger hypoperfused areas than in non-SIP patients, which may result in pathological damage to the brain that is responsible for the progression of stroke.