Galina Smushkin
Mayo Clinic
11 Papers
76 Citations
Galina Smushkin is an academic researcher from Mayo Clinic. The author has contributed to research in topics: Type 2 diabetes & Insulin. The author has an hindex of 7, co-authored 10 publications.
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Papers
Adult Height in Patients with Congenital Adrenal Hyperplasia: A Systematic Review and Metaanalysis
Kalpana Muthusamy,Mohamed B. Elamin,Galina Smushkin,Mohammad Hassan Murad,Julianna F. Lampropulos,Khalid B. Elamin,Nisrin O. Abu Elnour,Juan F. Gallegos-Orozco,Mitra M. Fatourechi,Neera Agrwal,Melanie A. Lane,Felipe N. Albuquerque,Patricia J. Erwin,Victor M. Montori +13 more
TL;DR: Evidence derived from observational studies suggests that the final height of CAH patients treated with glucocorticoids is lower than the population norm and is higher than expected given parental height.
Diabetes-Associated Common Genetic Variation and Its Association With GLP-1 Concentrations and Response to Exogenous GLP-1
Galina Smushkin,Matheni Sathananthan,Airani Sathananthan,Chiara Dalla Man,Francesco Micheletto,Alan R. Zinsmeister,Claudio Cobelli,Adrian Vella +7 more
TL;DR: In nondi diabetic subjects, diabetes-associated genetic variation does not alter GLP-1 concentrations after an oral challenge or its effect on insulin secretion, and in nondiabetic subjects, the associations of response with genotype are validated.
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Genetics of type 2 diabetes.
Galina Smushkin,Adrian Vella +1 more
TL;DR: The availability of large population-based cohorts and the ease with which large numbers of common genetic variants can be genotyped has enabled the discovery of multiple loci and pathways associated with type 2 diabetes.
Defects in GLP-1 Response to an Oral Challenge Do Not Play a Significant Role in the Pathogenesis of Prediabetes
Galina Smushkin,Airani Sathananthan,Chiara Dalla Man,Alan R. Zinsmeister,Michael Camilleri,Claudio Cobelli,Robert A. Rizza,Adrian Vella +7 more
TL;DR: The lack of association of GLP-1 concentrations with glucose tolerance status and with insulin secretion and action in a cohort encompassing the full spectrum of prediabetes strongly argues against a significant contribution of defects in GLP -1 secretion to the pathogenesis ofprediabetes.
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