Fengde Tian
Dalian University of Technology
13 Papers
35 Citations
Fengde Tian is an academic researcher from Dalian University of Technology. The author has contributed to research in topics: Femoral head & Medicine. The author has an hindex of 7, co-authored 13 publications.
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Papers
Treatment of early stage osteonecrosis of the femoral head with autologous implantation of bone marrow-derived and cultured mesenchymal stem cells
TL;DR: Ex vivo expansion of autologous BMMSCs can reliably provide a greater number of bone marrow mesenchymal stem cells for FH implantation and is safe and effective in delaying or avoiding FH collapse, which may necessitate total hip replacement.
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Omentin-1 prevents cartilage matrix destruction by regulating matrix metalloproteinases
TL;DR: Administration of omentin-1 abolished IL-1β-induced degradation of type II collagen and aggrecan, the two major extracellular matrix components in articular cartilage, in a dose-dependent manner and indicated that omentIn-1 may have a potential chondroprotective therapeutic capacity.
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Protective effects of Nebivolol against interleukin-1β (IL-1β)-induced type II collagen destruction mediated by matrix metalloproteinase-13 (MMP-13)
TL;DR: The results indicate that Nebivolol alleviated the increase in gene expression, protein expression, and activity of MMP-13 induced by IL-1β, and suggests that Nebvolol can potentially be applied in human OA treatment.
23
Combined vascularized iliac and greater trochanter graftings for reconstruction of the osteonecrosis femoral head with collapse: reports of three cases with 20 years follow-up.
TL;DR: Results show that the vascularized iliac and greater trochanter graftings may be a valuable option for reconstruction of the osteonecrosis of the femoral head with collapse in younger patients.
22
Omentin-1 promotes mitochondrial biogenesis via PGC1α-AMPK pathway in chondrocytes.
TL;DR: It is proved that omentin-1 has the potential to remedy chondrocyte damage in the prevention and treatment of osteoarthritis, and its action is dependent on the AMPK-PGC1α pathway.
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