Feng Ye
Nanjing Medical University
18 Papers
2 Citations
Feng Ye is an academic researcher from Nanjing Medical University. The author has contributed to research in topics: Biology & Helicobacter pylori. The author has an hindex of 6, co-authored 15 publications.
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Papers
Identification of the long non-coding RNA H19 in plasma as a novel biomarker for diagnosis of gastric cancer
TL;DR: Plasma H19 could serve as a potential biomarker for diagnosis of GC, in particular for early tumor screening and could be utilized to detect and monitor GC.
Deoxycholic acid induces gastric intestinal metaplasia by activating STAT3 signaling and disturbing gastric bile acids metabolism and microbiota
Duochen Jin,Keting Huang,Miao Xu,Hongjin Hua,Feng Ye,Jin Yan,Guoxin Zhang,Yun Wang +7 more
TL;DR: A BA-triggered TGR5/STAT3/KLF5 pathway in human gastric IM tissues was revealed and promoted proliferation and apoptotic resistance, upregulated proinflammatory cytokines and IM markers, and facilitated STAT3 phosphorylation, nuclear accumulation and DNA binding to the KLF5 promoter.
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A MDM2‐dependent positive‐feedback loop is involved in inhibition of miR‐375 and miR‐106b induced by Helicobacter pylori lipopolysaccharide
Feng Ye,Chun-Li Tang,Wei-jia Shi,Juan Qian,Shuping Xiao,Min Gu,Yini Dang,Jianping Liu,Yan Chen,Ruihua Shi,Guoxin Zhang +10 more
TL;DR: The results suggest that gastric epithelial cells treated with H. pylori LPS may be susceptible to JAK/STAT3 signal pathway activation via inhibition of miR‐375 and miR-106b.
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Eradication of Helicobacter pylori Infection Reduces the Incidence of Peptic Ulcer Disease in Patients using Nonsteroidal Anti-Inflammatory Drugs: A Meta-Analysis
TL;DR: This study aims to investigate the association between use of nonsteroidal anti‐inflammatory drugs (NSAID) and Helicobacter pylori infection, interactive effect of H. pylOR infection and NSAID use on the development of peptic ulcer disease (PUD), and the effect of NSAID eradication therapy on PUD development.
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Gastric cancer proliferation and invasion is reduced by macrocalyxin C via activation of the miR-212-3p/Sox6 Pathway.
TL;DR: It is concluded that macrocalyxin C may halt the proliferation of gastric malignancies through alteration of cell invasion, apoptosis, progression through the cell cycle and cell growth.
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