Feng He
Peking University
5 Papers
Feng He is an academic researcher from Peking University. The author has contributed to research in topics: microRNA & Cell cycle checkpoint. The author has an hindex of 3, co-authored 5 publications.
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Papers
The protective role of microRNA-21 against coxsackievirus B3 infection through targeting the MAP2K3/P38 MAPK signaling pathway
TL;DR: In this paper, microRNA-21 has potential inhibitory effect on the MAP2K3 which locates upstream of P38 MAPK and was upregulated in mouse hearts upon CVB3 infection.
Changes in N6-Methyladenosine Modification Modulate Diabetic Cardiomyopathy by Reducing Myocardial Fibrosis and Myocyte Hypertrophy.
TL;DR: In this article, the authors systematically profile global RNA N6-methyladenosine (m6A) modification patterns in a mouse model of diabetic cardiomyopathy (DCM).
Plasma interleukin-37 is increased and inhibits the production of inflammatory cytokines in peripheral blood mononuclear cells in systemic juvenile idiopathic arthritis patients
TL;DR: Levels of IL-37 were higher in sJIA patients, which were correlated with disease activity and sJia related inflammatory cytokines, suggesting thatIL-37 may have the potential role as a natural inhibitor for the pathogenesis and therapy of sJ IA.
A time-resolved proteomic analysis of transcription factors regulating adipogenesis of human adipose derived stem cells.
TL;DR: This study identified and verified three TFs (BATF3, MAFF and MXD4) as novel regulators of adipogenesis, whose over-expression could inhibit adipogenesis of hADSCs in vitro, and provided a valuable TFs resource to understand the complex process of adipesis.
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miR-140-5p could suppress tumor proliferation and progression by targeting TGFBRI/SMAD2/3 and IGF-1R/AKT signaling pathways in Wilms’ tumor
TL;DR: It is demonstrated that miRNA-140-5p participates in the progression of Wilms’ tumor by targeting the TGFBRI/SMAD2/3 and the IGF-1R/AKT signaling pathways.