Farida Emran
Harvard University
9 Papers
62 Citations
Farida Emran is an academic researcher from Harvard University. The author has contributed to research in topics: Zebrafish & Electroretinography. The author has an hindex of 6, co-authored 9 publications.
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Papers
A behavioral assay to measure responsiveness of zebrafish to changes in light intensities.
TL;DR: It is confirmed that the immediate behavioral responses to light-intensity changes require intact eyes by using the chokh (chk) mutants, which completely lack eyes from the earliest stages of development, which does not test the ability of zebrafish larvae to respond to changes in light intensities.
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OFF ganglion cells cannot drive the optokinetic reflex in zebrafish
TL;DR: It is concluded that the abnormal photoreceptor terminals in nrc mutants predominantly perturb the ON pathway and that the On pathway is necessary to drive the OKR in larval zebrafish.
140
Zebrafish larvae lose vision at night
TL;DR: It is shown that larval zebrafish essentially turn off their visual system at night when they are not active, a phenomenon that is largely circadian driven as fish show similar dramatic changes in visual responsiveness when maintained in continuous darkness, although light exposure at night partially restores the responses.
109
The bugeye mutant zebrafish exhibits visual deficits that arise with the onset of an enlarged eye phenotype.
TL;DR: The bugeye mutant provides a means of studying glaucoma-associated phenotypes in the zebrafish and suggests that there is a correlation between the size of the enlarged eye and the degree of OMR deficit.
Cellular expression of Smarca4 (Brg1)-regulated genes in zebrafish retinas.
Monica R. Hensley,Farida Emran,Sylvia Bonilla,Liyun Zhang,Wenxuan Zhong,Paul Grosu,John E. Dowling,Yuk Fai Leung,Yuk Fai Leung +8 more
TL;DR: This study has successfully investigated the expression pattern of 32 genes identified from the original factorial microarray analysis and demonstrated that the true discovery rate for identifying Smarca4-regulated retinal genes is 90.3%.