8 Papers
19 Citations
Farhan Malik is an academic researcher from University of Texas Health Science Center at Houston. The author has contributed to research in topics: Lung & Inflammation. The author has an hindex of 4, co-authored 7 publications.
Chat about Author
Papers
Effect of antigen sensitization and challenge on oscillatory mechanics of the lung and pulmonary inflammation in obese carboxypeptidase E-deficient mice
Paul H. Dahm,Jeremy B. Richards,Harry Karmouty-Quintana,Kevin R. Cromar,Sanjiv Sur,Roger E. Price,Farhan Malik,Chantal Y. Spencer,Ramon X. Barreno,S. Shahrukh Hashmi,Michael R. Blackburn,Ikram U. Haque,Richard A. Johnston +12 more
TL;DR: It is demonstrated that OVA sensitization and challenge enhance airway obstruction in obese mice regardless of the genetic basis of obesity, whereas the degree of OVA-induced pulmonary inflammation is dependent on the genetic modality of obesity induction.
15
Effect of antigen sensitization and challenge on oscillatory
Paul H. Dahm,Jeremy B. Richards,Harry Karmouty-Quintana,Kevin R. Cromar,Farhan Malik,Chantal Y. Spencer,Ramon X. Barreno,S Syed,Michael R. Blackburn,Ikram U. Haque,Richard A. Johnston +10 more
- 01 Jan 2014
TL;DR: In this paper, the authors examined indices of these sequelae in mice obese due to a genetic deficiency in carboxypeptidase E, an enzyme that processes pro-hormones and pro-neuropeptides involved in satiety and energy expenditure.
14
Resistin deficiency in mice has no effect on pulmonary responses induced by acute ozone exposure.
Shehla S. Razvi,Jeremy B. Richards,Farhan Malik,Kevin R. Cromar,Roger E. Price,Cynthia S. Bell,Tingting Weng,Constance L. Atkins,Chantal Y. Spencer,Katherine J. Cockerill,Amy L. Alexander,Michael R. Blackburn,Joseph L. Alcorn,Ikram U. Haque,Richard A. Johnston +14 more
TL;DR: It is demonstrated that murine resistin is increased in the lungs of wild-type mice following acute O3 exposure but does not promote O3-induced lung pathology.
11
Chemokine (C-C Motif) Receptor-Like 2 is not essential for lung injury, lung inflammation, or airway hyperresponsiveness induced by acute exposure to ozone.
Farhan Malik,Kevin R. Cromar,Constance L. Atkins,Roger E. Price,William T. Jackson,Saad R. Siddiqui,Chantal Y. Spencer,Nicholas C. Mitchell,Ikram U. Haque,Richard A. Johnston +9 more
TL;DR: It is demonstrated that Ccrl2 modulates chemerin levels in the epithelial lining fluid of the lungs but does not contribute to the development of O3‐induced lung pathology.
Plasminogen activator inhibitor‐1 does not contribute to the pulmonary pathology induced by acute exposure to ozone
Hamza S. Elkhidir,Jeremy B. Richards,Kevin R. Cromar,Cynthia S. Bell,Roger E. Price,Constance L. Atkins,Chantal Y. Spencer,Farhan Malik,Amy L. Alexander,Katherine J. Cockerill,Ikram U. Haque,Richard A. Johnston +11 more
TL;DR: Following acute exposure to O3, PAI‐1 neither regulates pulmonary expression of IL‐6 and KC nor functionally contributes to any of the pulmonary pathological sequelae that arise from the noxious effects of inhaled O3.
4