F. Manz
Boston Children's Hospital
6 Papers
48 Citations
F. Manz is an academic researcher from Boston Children's Hospital. The author has contributed to research in topics: Net acid excretion & Metabolic acidosis. The author has an hindex of 6, co-authored 6 publications.
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Papers
Alkali therapy versus sodium chloride supplement in low birthweight infants with incipient late metabolic acidosis.
TL;DR: In patients with incipient late metabolic acidosis, NaCl therapy is not as beneficial as NaHCO3 therapy, and 27 patients on NaH CO3 therapy showed an increase in urine pH values, combined with a relatively high gain in body weight and a tendency to increased N‐assimilation.
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Suboptimal mineral composition of cow's milk formulas: a risk factor for the development of late metabolic acidosis.
TL;DR: Suboptimal mineral composition of the formulas turned out to be an important risk factor for the development of late metabolic acidosis in a term baby boy with renal tubular acidosis type 4 who received two cow's milk formulas in succession.
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Increased Renal Net Acid Excretion in Prematures below 1,600 g Body Weight Compared with Prematures and Small-for-Date Newborns above 2,100 g on Alimentation with a Commercial Preterm Formula
TL;DR: Urinary ionogram data of these 2 groups of infants show that the increased renal net acid excretion of the smaller prematures is the result of a lower urinaryexcretion of sodium, potassium and chloride, due to a higher daily weight gain, probably a higher retention of these minerals, and a higher urinary phosphorus excretion probably due to an age-specific lower intestinal calcium absorption, and therefore a lower rate of calcium and phosphorus retention.
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Effect of calcium supplementation on calcium and phosphorus balance and renal net acid excretion in preterm infants fed a standard formula
F. Manz,L Diekmann,GJ Stock +2 more
TL;DR: Increased Ca intake given to optimize the Ca:P ratio improves mineral retention in preterm infants fed a standard formula, and balance studies showed increased net intestinal Ca absorption during supplementation as well as improved Ca and P retention.
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Low renal net acid excretion, high calciuria and biochemical signs of sodium deficiency in low-birth-weight infants fed a new low-phosphorus formula.
TL;DR: Low urinary Na excretion and increased urinary excretion of aldosterone‐18‐glucuronide indicated biochemical evidence of Na deficiency secondary to low Na intake and a high weight gain, which should be higher than in mature human milk because of the often relatively higher weight gain.
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