Eshcar Meisel
Tel Aviv University
11 Papers
5 Citations
Eshcar Meisel is an academic researcher from Tel Aviv University. The author has contributed to research in topics: Gating & Medicine. The author has an hindex of 4, co-authored 8 publications. Previous affiliations of Eshcar Meisel include Sheba Medical Center.
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Papers
The Prognostic Role of Neutrophil-to-Lymphocyte Ratio in Patients Hospitalized with Acute Pulmonary Embolism.
Orly Efros,Orly Efros,Tal Beit Halevi,Eshcar Meisel,Eshcar Meisel,Shelly Soffer,Shelly Soffer,Noam Barda,Noam Barda,Omri Cohen,Gili Kenet,Gili Kenet,Aharon Lubetsky,Aharon Lubetsky +13 more
TL;DR: In this article, the prognostic value of neutrophil-to-lymphocyte ratio (NLR) in patients hospitalized with acute pulmonary embolism by investigating its association with mortality in a large-scale population diagnosed with acute PE.
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Andersen-Tawil Syndrome Is Associated With Impaired PIP2 Regulation of the Potassium Channel Kir2.1.
Reem Handklo-Jamal,Eshcar Meisel,Eshcar Meisel,Daniel Yakubovich,Leonid Vysochek,Roy Beinart,Roy Beinart,Michael Glikson,Michael Glikson,Julie R. McMullen,Nathan Dascal,Eyal Nof,Eyal Nof,Shimrit Oz,Shimrit Oz +14 more
TL;DR: An impaired function and a dominant-negative effect of the Kir2.1 mutant dimers current compared to the WT dimer are suggested, and BGP-15, a drug that has been demonstrated to have an anti-arrhythmic effect in mice, stabilized the Kir1.1 current amplitude.
Risk factors and mortality in patients with pneumonia and elevated troponin levels.
Orly Efros,Shelly Soffer,Avshalom Leibowitz,Avshalom Leibowitz,Alexander Fardman,Alexander Fardman,Robert Klempfner,Robert Klempfner,Eshcar Meisel,Eshcar Meisel,Ehud Grossman,Ehud Grossman +11 more
TL;DR: In hospitalized patients with pneumonia elevated troponin levels have a major impact on prognosis, and trop onin levels may be used as another tool of risk stratification for patients hospitalized with pneumonia.
Mutations in NaV1.5 Reveal Calcium-Calmodulin Regulation of Sodium Channel.
Eyal Nof,Eyal Nof,Leonid Vysochek,Eshcar Meisel,Eshcar Meisel,Elena Burashnikov,Charles Antzelevitch,Charles Antzelevitch,Jérôme Clatot,Roy Beinart,Roy Beinart,David Luria,Michael Glikson,Michael Glikson,Shimrit Oz +14 more
TL;DR: The results highlight the role of the NaV1.5 DIII-IV linker in the CaM-binding complex and channel function, and suggest that the Ca2+-sensing machinery of NaV 1.5 involves NaVβ1.
Inactivation gating of Kv7.1 channels does not involve concerted cooperative subunit interactions.
TL;DR: Results indicate that Kv7.1 inactivation gating is not compatible with a concerted cooperative process, and suggests that like for activation gating, Kv 7.1 slow inactivate gATING is not a concerted process.
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