Erez Geron
Weizmann Institute of Science
10 Papers
77 Citations
Erez Geron is an academic researcher from Weizmann Institute of Science. The author has contributed to research in topics: Secretory Vesicle & Apical membrane. The author has an hindex of 4, co-authored 7 publications.
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Papers
Lymphocyte Crawling and Transendothelial Migration Require Chemokine Triggering of High-Affinity LFA-1 Integrin
Ziv Shulman,Vera Shinder,Eugenia Klein,Valentin Grabovsky,Orna Yeger,Erez Geron,Alessio Montresor,Matteo Bolomini-Vittori,Sara W. Feigelson,Tomas Kirchhausen,Carlo Laudanna,Guy Shakhar,Ronen Alon +12 more
TL;DR: It is proposed that shear forces exerted on HA-LFA-1 trigger adhesive and invasive filopodia at apical endothelial surfaces and thereby promote lymphocyte crawling and probing for TEM sites.
273
The Edges of Pancreatic Islet β Cells Constitute Adhesive and Signaling Microdomains
TL;DR: Murine β cell edges are identified, the sites at which adjacent cell faces meet at a sharp angle, as surface microdomains of cell-cell adhesion and signaling, and the spatial architecture of their edges may prove fundamental for coordinating physiological insulin secretion.
61
Directing exocrine secretory vesicles to the apical membrane by actin cables generated by the formin mDia1
TL;DR: The role of Diaphanous formins at the final stages of secretion appears to be highly conserved, with similar apical secretory tracks found in the submandibular salivary glands of Drosophila.
50
Suppression of experimental autoimmune myasthenia gravis by inhibiting the signaling between IFN-γ inducible protein 10 (IP-10) and its receptor CXCR3
Tali Feferman,Revital Aricha,Keren Mizrachi,Keren Mizrachi,Erez Geron,Ronen Alon,Miriam C. Souroujon,Miriam C. Souroujon,Sara Fuchs +8 more
TL;DR: The potential of modulating rat EAMG by interference in CXCR3/IP-10 signaling can be considered as a potential treatment modality for MG.
28
Generalized extinction of fear memory depends on co-allocation of synaptic plasticity in dendrites
TL;DR: In this article , the authors show that fear conditioning with two different auditory stimuli (CS) and footshocks (US) induces dendritic spine elimination mainly on different Dendritic branches of layer 5 pyramidal neurons in the mouse motor cortex.