Endre Anderssen
Harvard University
50 Papers
297 Citations
Endre Anderssen is an academic researcher from Harvard University. The author has contributed to research in topics: Medicine & Cancer. The author has an hindex of 24, co-authored 46 publications. Previous affiliations of Endre Anderssen include Weatherford International & Norwegian University of Science and Technology.
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Papers
A Molecular Roadmap of Reprogramming Somatic Cells into iPS Cells
Jose M. Polo,Endre Anderssen,Endre Anderssen,Ryan M. Walsh,Benjamin A. Schwarz,Christian M. Nefzger,Sue Mei Lim,Marti Borkent,Marti Borkent,Effie Apostolou,Sara Alaei,Jennifer Cloutier,Ori Bar-Nur,Sihem Cheloufi,Matthias Stadtfeld,Maria E. Figueroa,Daisy A. Robinton,Sridaran Natesan,Ari Melnick,Jinfang Zhu,Sridhar Ramaswamy,Sridhar Ramaswamy,Konrad Hochedlinger,Konrad Hochedlinger +23 more
TL;DR: This integrative analysis allowed us to identify genes that act as roadblocks during reprogramming and surface markers that further enrich for cells prone to forming iPSCs, and offer new mechanistic insights into the nature and sequence of molecular events inherent to cellular reprograming.
891
Regression of a data matrix on descriptors of both its rows and of its columns via latent variables: L-PLSR
Harald Martens,Harald Martens,Endre Anderssen,Arnar Flatberg,Lars Gidskehaug,Martin Høy,Frank Westad,Anette Kistrup Thybo,Magni Martens +8 more
TL;DR: The L-PLSR is applied to the analysis of consumer liking data Y of six products assessed by 125 persons, in light of 10 other product descriptors X and 15 other person descriptors Z.
91
Phylogenetic backgrounds and virulence profiles of atypical enteropathogenic Escherichia coli strains from a case-control study using multilocus sequence typing and DNA microarray analysis.
TL;DR: The atypical EPEC strains were heterogeneous both phylogenetically and by virulence profile, and Phylogenetic ancestry was less useful as a predictor of diarrhea than were specific virulence genes.
85
RBF binding to both canonical E2F targets and noncanonical targets depends on functional dE2F/dDP complexes.
TL;DR: Results suggest that E2F/DP complexes are essential for all genomic targeting of RBF1.
62
A shared role for RBF1 and dCAP-D3 in the regulation of transcription with consequences for innate immunity.
Michelle S. Longworth,James A. Walker,Endre Anderssen,Nam-Sung Moon,Andrew B. Gladden,Margarete M. S. Heck,Sridhar Ramaswamy,Nicholas J. Dyson +7 more
TL;DR: It is shown that the absence of RBF1 and dCAP-D3 alters the expression of many of the same genes in larvae and adult flies, and it is revealed that RBF 1 and d Caps 3 are needed in fat body cells to activate transcription of clusters of antimicrobial peptide genes.