Enamul Haque
Chittaranjan National Cancer Institute
9 Papers
144 Citations
Enamul Haque is an academic researcher from Chittaranjan National Cancer Institute. The author has contributed to research in topics: Immune system & Downregulation and upregulation. The author has an hindex of 9, co-authored 9 publications.
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Papers
Neem (Azadirachta indica) leaf preparation induces prophylactic growth inhibition of murine Ehrlich carcinoma in Swiss and C57BL/6 mice by activation of NK cells and NK-T cells
Enamul Haque,Rathindranath Baral +1 more
TL;DR: NLP-activated NK and NK-T cells in mice may regulate tumor cell cytotoxicity by enhancing the secretion of different cytotoxic cytokines.
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Neem leaf preparation induces apoptosis of tumor cells by releasing cytotoxic cytokines from human peripheral blood mononuclear cells.
TL;DR: Observations suggested that NLP could induce tumor cellular apoptosis by releasing cytotoxic cytokines from human PBMC.
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Pretreatment with neem (Azadirachta indica) leaf preparation in swiss mice diminishes leukopenia and enhances the antitumor activity of cyclophosphamide
TL;DR: NLP would be an effective tool to reduce CYP‐induced hematological complications and in vitro tumor cell cytotoxicity by peripheral blood mononuclear cells from CYP treated mice in either normal or tumor bearing conditions.
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Induction of type 1 cytokines during neem leaf glycoprotein assisted carcinoembryonic antigen vaccination is associated with nitric oxide production.
Koustav Sarkar,Anamika Bose,Enamul Haque,Krishnendu Chakraborty,Tathagata Chakraborty,Shyamal Goswami,Diptendu Ghosh,Rathindranath Baral +7 more
TL;DR: Results clearly demonstrated the interdependence of two anti-tumor immune functions, namely, NO production and generation of type 1 immune response.
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Neem (Azadirachta indica) leaf preparation prevents leukocyte apoptosis mediated by cisplatin plus 5-fluorouracil treatment in Swiss mice.
TL;DR: Significant down-regulation of leukocyte apoptosis was noted in mice pretreated with NLP or granulocyte colony stimulating factor (GCSF) during cis + 5-FU therapy, suggesting enhanced cytotoxicity may be associated with N LP-induced increase of the cytotoxic T and NK cell pool.
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