E. Pedernera
National Autonomous University of Mexico
40 Papers
181 Citations
E. Pedernera is an academic researcher from National Autonomous University of Mexico. The author has contributed to research in topics: Ovary & Embryo. The author has an hindex of 14, co-authored 40 publications.
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Papers
The P450 aromatase (P450 arom) gene is asymmetrically expressed in a critical period for gonadal sexual differentiation in the chick.
TL;DR: It is proposed that there is a gene in birds that is asymmetrically expressed in the undifferentiated stage of the female and the male gonad that could promote P450 arom gene expression, increasing estrogen production, which in turn could induce ovarian cortex proliferation and expression of other structural estrogen-regulated genes involved in ovarian sexual determination.
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Heterogeneity of growth hormone immunoreactivity in lymphoid tissues and changes during ontogeny in domestic fowl
Maricela Luna,N. Barraza,Laura C. Berumen,Martha Carranza,E. Pedernera,Steve Harvey,Carlos Arámburo +6 more
TL;DR: These results demonstrate age-related and tissue-specific changes in the content and composition of GH in immune tissues of the chicken, in which GH is likely to be an autocrine or paracrine regulator.
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Proliferative effect in vitro of follicle-stimulating hormone on the left ovary of the chick embryo.
TL;DR: A dose-dependent stimulation of DNA synthesis by hFSH in the inmature ovary of the 18-day-old chick embryo is demonstrated to demonstrate the proliferative effect of hF SH on cellular proliferation in the chick embryo ovary.
18
Progesterone reduces cell survival in primary cultures of endometrioid ovarian cancer
TL;DR: This study supports the importance of progesterone and the presence of progestersone receptor in the reduction of ovarian cancer progression in the endometrioid ovarian carcinoma.
Androgen receptor is expressed in mouse cardiomyocytes at prenatal and early postnatal developmental stages
TL;DR: Evidence that androgen receptor is expressed from prenatal stages in mouse heart is provided, supporting the proposition that androgens could be involved in mammalian heart development.