E. L. White
Monash University
11 Papers
165 Citations
E. L. White is an academic researcher from Monash University. The author has contributed to research in topics: Familial dysalbuminemic hyperthyroxinemia & Albumin. The author has an hindex of 10, co-authored 11 publications. Previous affiliations of E. L. White include Alfred Hospital.
Chat about Author
Papers
Familial euthyroid thyroxine excess: an appropriate response to abnormal thyroxine binding associated with albumin.
TL;DR: It is suggested that T4 excess is an appropriate response to abnormal T4 binding so as to maintain normal free T4, and the excess bound T4 is associated with a normal quantity of albumin.
121
Limitations of a new free thyroxine assay (amerlex® free t4)
TL;DR: It appears that Amerlex® Free T4 is influenced by lower‐affinity, high‐capacity T4 binding sites in serum, so that apparent free T4 concentration may vary with changes in the concentration of such sites.
113
Evaluation of Free Thyroxine Methods in the Presence of Iodothyronine-Binding Autoantibodies*
Paolo Beck-Peccoz,P.B. Romelli,M.G. Cattaneo,Giovanni Faglia,E. L. White,John W. Barlow,Jan R. Stockigt +6 more
TL;DR: It is suggested that FT4 methods are valid in patients with circulating iodothyronine-binding immunoglobulins only if the free hormone fraction is physically separated from serum binding proteins before the assay procedure.
72
A Pituitary Tumor Producing High Molecular Weight Adrenocorticotropin-Related Peptides: Clinical and Cell Culture Studies*
Peter J. Fuller,Alan T.W. Lim,John W. Barlow,E. L. White,B. A. K. Khalid,David L. Copolov,Steven Lolait,John W. Funder,Jan R. Stockigt +8 more
TL;DR: Using this cell line, it is demonstrated that the biosynthesis of POMC, its pattern of processing, and the release of Pombelanocortin/ir-beta EP/IR-ACTH in vitro were consistent with the in vivo evidence of autonomous secretion and abnormal processing of PomC by this pituitary tumor.
53
Specific methods to identify plasma binding abnormalities in euthyroid hyperthyroxinemia.
TL;DR: Use of these techniques rules out the known binding abnormalities in hyperthyroxinemic patients and may make the diagnosis of generalized hormone resistance more specific and help identify individuals and families at risk of misdiagnosis by standard methods.
32