E. Agneter
University of Vienna
18 Papers
284 Citations
E. Agneter is an academic researcher from University of Vienna. The author has contributed to research in topics: Agonist & Microdialysis. The author has an hindex of 13, co-authored 18 publications.
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Papers
Characterization of peripheral-compartment kinetics of antibiotics by in vivo microdialysis in humans.
Markus Müller,O Haag,T. Burgdorff,A. Georgopoulos,W Weninger,Burkhard Jansen,Gerold Stanek,Hubert Pehamberger,E. Agneter,Hans-Georg Eichler +9 more
TL;DR: It is demonstrated, by means of microdialysis, that the concept of pharmacokinetic/pharmacodynamic surrogate markers for evaluation of antibiotic regimens originally developed for serum pharmacokinetics can be extended to peripheral-tissue pharmacokinetically.
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Sustained dopamine release induced by secretoneurin in the striatum of the rat: a microdialysis study.
E. Agneter,Harald H. Sitte,S. Stöckl-Hiesleitner,Reiner Fischer-Colbrie,Hans Winkler,Ernst A. Singer +5 more
TL;DR: It is established that SN has a pronounced effect on DA release under in vivo conditions.
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Comparison of the Effects of Ketamine and Memantine on Prolactin and Cortisol Release in Men: A Randomized, Double-blind, Placebo-controlled Trial
Nicole Hergovich,Ernst A. Singer,E. Agneter,Hans Georg Eichler,Ursula Graselli,Christian Simhandl,Bernd Jilma +6 more
TL;DR: Ketamine increased serum prolactin and cortisol levels, whereas memantine and placebo did not affect hormone levels, and further studies are needed to define whether higher doses of memantine or other NMDA antagonists can induce hormone release.
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Induction by low Na+ or Cl− of cocaine sensitive carrier‐mediated efflux of amines from cells transfected with the cloned human catecholamine transporters†
TL;DR: Cloned catecholamine transporters display carrier‐mediated efflux of amines if challenged by lowering the extracellular Na+ or Cl−, whilst retaining their pharmacological profile.
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Amphetamine reverses or blocks the operation of the human noradrenaline transporter depending on its concentration: superfusion studies on transfected cells.
TL;DR: It is concluded that amphetamine can induce noradrenaline transporter mediated release only at high nanomolar to low micromolar concentrations and that the releasing action of amphetamine, like that of cocaine, is dependent on a vesicular pool of noradRenaline.
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